Axonal damage in multiple sclerosis patients with high versus low expanded disability status scale score

Can J Neurol Sci. 2004 May;31(2):225-8. doi: 10.1017/s0317167100053877.

Abstract

Background: The pathophysiological basis for differences in disability in patients with multiple sclerosis is unclear.

Methods: We used magnetic resonance imaging to examine whether differences in disability in cohorts of multiple sclerosis patients with similar T2-weighted lesion volume and disease duration were associated with a more destructive disease process in the more disabled patients.

Results: The benign and severely disabled groups had similar brain atrophy metrics and similar decreases of the neuronal marker, N-acetylaspartate, in the normal appearing white matter of the cerebrum on magnetic resonance spectroscopy examination in vivo. The severely disabled cohort had more spinal cord atrophy.

Conclusion: The dissociation of spinal cord atrophy and cerebral atrophy between these two groups suggests that the difference between the more benign and more disabled groups cannot be explained by a more aggressive pathological process that is affecting the entire neuroaxis in a homogeneous fashion.

Publication types

  • Clinical Trial
  • Comparative Study
  • Controlled Clinical Trial

MeSH terms

  • Adult
  • Aspartic Acid / analogs & derivatives*
  • Aspartic Acid / metabolism
  • Atrophy
  • Axons / pathology*
  • Biomarkers
  • Brain / metabolism
  • Brain / pathology*
  • Disability Evaluation*
  • Humans
  • Magnetic Resonance Imaging
  • Matched-Pair Analysis
  • Middle Aged
  • Multiple Sclerosis / classification
  • Multiple Sclerosis / diagnosis*
  • Multiple Sclerosis / physiopathology
  • Retrospective Studies
  • Severity of Illness Index
  • Spinal Cord / pathology*

Substances

  • Biomarkers
  • Aspartic Acid
  • N-acetylaspartate