Specific inhibition effects of N-pentafluorobenzyl-1-deoxynojirimycin on human CD4+ T cells

Bioorg Med Chem Lett. 2004 Jul 16;14(14):3789-92. doi: 10.1016/j.bmcl.2004.04.092.

Abstract

We first synthesized N-pentafluorobenzyl-1-deoxynojirimycin (5F-DNM), one new derivative of 1-deoxynojirimycin (DNM). Effects on human peripheral blood mononuclear cells (PMBC) and secretion of cytokines from human PBMC by 5F-DNM were investigated. It was first found that 5F-DNM remarkably inhibited the secretion of interleukin-4 (IL-4) and had a specific inhibition on the expression of CD4 molecules. 5F-DNM, much less toxic than cyclosporin A, might be used as a new candidate of immunosuppressant for specifically treating Th2-mediated immune diseases.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 1-Deoxynojirimycin / analogs & derivatives*
  • 1-Deoxynojirimycin / chemical synthesis
  • 1-Deoxynojirimycin / pharmacology*
  • Benzyl Compounds / chemical synthesis
  • Benzyl Compounds / pharmacology*
  • CD4-Positive T-Lymphocytes / cytology
  • CD4-Positive T-Lymphocytes / drug effects*
  • CD4-Positive T-Lymphocytes / metabolism
  • Cyclosporine / therapeutic use
  • Cytokines / metabolism
  • Humans
  • Immunosuppressive Agents / chemical synthesis
  • Immunosuppressive Agents / pharmacology*
  • Interleukin-4 / metabolism
  • Leukocytes, Mononuclear / drug effects
  • Leukocytes, Mononuclear / metabolism
  • Th2 Cells / drug effects
  • Th2 Cells / immunology
  • Th2 Cells / metabolism

Substances

  • Benzyl Compounds
  • Cytokines
  • Immunosuppressive Agents
  • N-pentafluorobenzyl-1-deoxynojirimycin
  • 1-Deoxynojirimycin
  • Interleukin-4
  • Cyclosporine