Intramembrane charge movement associated with endogenous K+ channel activity in HEK-293 cells

Cell Mol Neurobiol. 2004 Jun;24(3):317-30. doi: 10.1023/b:cemn.0000022765.52109.26.

Abstract

1. The use of molecular biology in combination with electrophysiology in the HEK-293 cell line has given fascinating insights into neuronal ion channel function. Nevertheless, to fully understand the properties of channels exogenously expressed in these cells, a detailed evaluation of endogenous channels is indispensable. 2. Previous studies have shown the expression of endogenous voltage-gated K+, Ca2+, and Cl- channels and this predicts that changes in membrane potential will cause intramembrane charge movement, though this gating charge translocation remain undefined. Here, we confirm this prediction by performing patch-clamp experiments to record ionic and gating currents. Our data show that HEK-293 cells express at least two types of K+-selective endogenous channels which sustain the majority of the ionic current, and exclude a significant contribution from Ca2+ and Cl- channels to the whole-cell current. 3. Gating currents were unambiguously resolved after ionic current blockade enabling this first report of intramembrane charge movement in HEK-293 cells arising entirely from endogenous K+ channel activity, and providing valuable information concerning the activation mechanism of voltage-gated K+ channels in these cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Calcium Channels / metabolism
  • Calcium Chloride / pharmacology
  • Cell Line
  • Cell Membrane / drug effects
  • Cell Membrane / metabolism*
  • Cesium / pharmacology
  • Chloride Channels / metabolism
  • Humans
  • Ion Channel Gating / drug effects
  • Ion Channel Gating / physiology*
  • Ions / metabolism*
  • Kidney / cytology
  • Kidney / drug effects
  • Kidney / metabolism*
  • Membrane Potentials / drug effects
  • Membrane Potentials / physiology
  • Niflumic Acid / pharmacology
  • Patch-Clamp Techniques
  • Potassium Channel Blockers / pharmacology
  • Potassium Channels, Voltage-Gated / antagonists & inhibitors
  • Potassium Channels, Voltage-Gated / metabolism*
  • Potassium Chloride / pharmacology
  • Sodium Chloride / pharmacology
  • Tetraethylammonium / pharmacology

Substances

  • Calcium Channels
  • Chloride Channels
  • Ions
  • Potassium Channel Blockers
  • Potassium Channels, Voltage-Gated
  • Cesium
  • Sodium Chloride
  • Niflumic Acid
  • Tetraethylammonium
  • Potassium Chloride
  • Calcium Chloride