Pharmacokinetics and pharmacodynamics of terbogrel, a combined thromboxane A2 receptor and synthase inhibitor, in healthy subjects

Br J Clin Pharmacol. 2004 Jul;58(1):40-51. doi: 10.1111/j.1365-2125.2004.02083.x.

Abstract

Aims: To characterize the pharmacokinetics of terbogrel, a new combined thromboxane A2 (TxA2) receptor and synthase inhibitor, in healthy human subjects after single or multiple oral administration.

Methods: Forty-eight healthy male subjects received a single oral dose (10, 25, 50, 100, 150 or 200 mg) of terbogrel or placebo and 32 different subjects received one of the following treatments twice daily for 7 days: 50, 100 or 150 mg terbogrel, placebo, or once-a-day 330 mg acetylsalicylic acid.

Results: Terbogrel was well tolerated without obvious adverse effects following either a single oral dose or administration over 7 days. Plasma drug concentrations were dose-linear and there was no accumulation over 7 days. There was a dose-dependent blockade of TxA2 receptors and of inhibition of thromboxane synthase activity with values for IC50 of 12 ng ml(-1) and 6.7 ng ml(-1), respectively. At the highest dose tested (150 mg) there was almost complete inhibition of thomboxane synthase and thromboxane receptor occupancy. Even at trough concentrations, receptor occupancy remained above 80% and thromboxane synthase was still completely inhibited. These two activities were associated with a dose-dependent inhibition of platelet aggregation (>80% at the 150 mg dose of terbogrel) and enhanced prostacyclin production.

Conclusions: Terbogrel is a potent agent having two distinct, complimentary pharmacodynamic actions, namely inhibition of thromboxane synthase and antagonism of the TxA2 receptor. The antithrombotic effect of terbogrel was dose-dependent and was associated with enhanced prostacyclin production. Terbogrel is an attractive candidate for long-term antithrombotic therapy.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial

MeSH terms

  • 6-Ketoprostaglandin F1 alpha / metabolism
  • Adult
  • Aspirin / administration & dosage
  • Bleeding Time
  • Dose-Response Relationship, Drug
  • Double-Blind Method
  • Humans
  • Male
  • Platelet Aggregation / drug effects
  • Pyridines / pharmacokinetics*
  • Pyridines / pharmacology
  • Thromboxane A2 / antagonists & inhibitors*
  • Thromboxane-A Synthase / antagonists & inhibitors*

Substances

  • Pyridines
  • Thromboxane A2
  • 6-Ketoprostaglandin F1 alpha
  • terbogrel
  • Thromboxane-A Synthase
  • Aspirin