Transmission of HIV-1 by primary human uterine epithelial cells and stromal fibroblasts

J Infect Dis. 2004 Jul 15;190(2):236-45. doi: 10.1086/421910. Epub 2004 Jun 11.

Abstract

Women can become infected with human immunodeficiency virus type 1 (HIV-1) after the heterosexual transmission of virus from an infected male partner. To understand the events that result in transmission of HIV-1 across the female reproductive tract, we characterized the life-cycle events of HIV-1 in primary cultures of human uterine epithelial cells and stromal fibroblasts. Epithelial cells and stromal fibroblasts released virus particles after exposure to either X4- or R5-tropic strains of HIV-1. Virus released by these cells was able to infect CD4(+) T cells. When exposed to an X4-tropic strain of HIV-1, these cells supported HIV-1 reverse transcription, integration, and viral DNA transcription. When exposed to an R5-tropic strain, however, these cells released unmodified virus. These data suggest that uterine cells are targets for productive infection with X4-tropic strains and release unmodified R5-tropic viruses that would then be able to infect submucosal target cells, including T cells and macrophages.

Publication types

  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • CD4-Positive T-Lymphocytes / virology
  • Cells, Cultured
  • Epithelial Cells / virology*
  • Female
  • Fibroblasts / virology*
  • HIV Core Protein p24 / analysis
  • HIV Infections / transmission*
  • HIV-1 / pathogenicity*
  • HIV-1 / physiology
  • Humans
  • Receptors, CCR5 / metabolism
  • Receptors, CXCR4 / biosynthesis
  • Receptors, CXCR4 / metabolism
  • Receptors, HIV / metabolism
  • Stromal Cells / virology
  • Transcription, Genetic
  • Uterus / cytology*
  • Uterus / virology*
  • Virus Integration
  • Virus Replication

Substances

  • HIV Core Protein p24
  • Receptors, CCR5
  • Receptors, CXCR4
  • Receptors, HIV