Abstract
Exercise training instituted after myocardial infarction improves many steps involved in cardiac excitation-contraction coupling. Focusing on Na/Caexchange, current controversies regarding whether it mediates Cainflux during an action potential, whether it is increased or decreased in disease models, whether protein kinase A alters its activity, and whether exercise training affects its function are reviewed. Finally, a novel target for exercise training in the heart is suggested.
Copyright 2004 American College of Sports Medicine
Publication types
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Research Support, Non-U.S. Gov't
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Research Support, U.S. Gov't, P.H.S.
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Review
MeSH terms
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Animals
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Calcium / metabolism
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Calcium / pharmacology
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Cell Size / drug effects
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Cyclic AMP-Dependent Protein Kinases / metabolism
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Dose-Response Relationship, Drug
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Exercise / physiology*
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Humans
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Myocardial Infarction / metabolism
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Myocardial Infarction / physiopathology*
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Myocytes, Cardiac / cytology
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Myocytes, Cardiac / drug effects
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Myocytes, Cardiac / physiology
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Physical Conditioning, Animal / physiology
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Rats
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Sodium-Calcium Exchanger / metabolism*
Substances
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Sodium-Calcium Exchanger
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Cyclic AMP-Dependent Protein Kinases
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Calcium