Effects of different hypocaloric diets on protein secretion from adipose tissue of obese women

Diabetes. 2004 Aug;53(8):1966-71. doi: 10.2337/diabetes.53.8.1966.

Abstract

Little is known about common factors (e.g., macronutrients and energy supply) regulating the protein secretory function of adipose tissue. We therefore compared the effects of randomly assigned 10-week hypoenergetic (-600 kcal/day) diets with moderate-fat/moderate-carbohydrate or low-fat/high-carbohydrate content on circulating levels and production of proteins (using radioimmunoassays and enzyme-linked immunosorbent assays) from subcutaneous adipose tissue in 40 obese but otherwise healthy women. Similar results were obtained by the two diets. Body weight decreased by approximately 7.5%. The secretion rate of leptin decreased by approximately 40%, as did that of tumor necrosis factor-alpha (TNF-alpha), and interleukin (IL)-6 and -8 decreased by 25-30%, whereas the secretion of plasminogen activator inhibitor 1 (PAI-1) and adiponectin did not show any changes. Regarding mRNA expression (by real-time PCR), only that of leptin and IL-6 decreased significantly. Circulating levels of leptin and PAI-1 decreased by 30 and 40%, respectively, but there were only minor changes in circulating TNF-alpha, IL-6, or adiponectin. In conclusion, moderate caloric restriction but not macronutrient composition influences the production and secretion of adipose tissue-derived proteins during weight reduction, leptin being the most sensitive and adiponectin and PAI-1 the least sensitive.

Publication types

  • Clinical Trial
  • Randomized Controlled Trial
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adipose Tissue / anatomy & histology
  • Adipose Tissue / metabolism*
  • Adult
  • Body Mass Index
  • Diet, Reducing*
  • Female
  • Humans
  • Interleukin-6 / genetics
  • Interleukin-8 / genetics
  • Leptin / genetics
  • Obesity / physiopathology*
  • Patient Compliance
  • Plasminogen Activator Inhibitor 1 / genetics
  • Proteins / genetics
  • Proteins / metabolism*
  • RNA, Messenger / genetics
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Interleukin-6
  • Interleukin-8
  • Leptin
  • Plasminogen Activator Inhibitor 1
  • Proteins
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha