IL-5 links adaptive and natural immunity in reducing atherosclerotic disease

J Clin Invest. 2004 Aug;114(3):317-9. doi: 10.1172/JCI22561.

Abstract

Oxidized LDL induces changes in several facets of the immune system, although the relationships between these facets and their contributions to atherogenesis have yet to be fully elucidated. A report in this issue of the JCI provides a novel demonstration of the adaptive immune system influencing the production of natural antibodies. The results demonstrate that injection of malondialdehyde-modified LDL promotes a Th2 response that in turn increases the titers of the natural antibody T15/EO6, which recognizes the oxidized phospholipid POVPC. Atherosclerotic lesion size in LDL receptor-deficient mice is reduced as a consequence of the increase in natural antibody titers, and IL-5 is identified as the link between the adaptive and natural immune systems.

Publication types

  • Comment
  • Review

MeSH terms

  • Animals
  • Antibody Formation / immunology*
  • Apolipoproteins E / deficiency
  • Arteriosclerosis / prevention & control*
  • Disease Models, Animal
  • Gene Expression Regulation / immunology*
  • Humans
  • Immunity, Innate*
  • Interleukin-5 / genetics
  • Interleukin-5 / metabolism*
  • Lipoproteins, LDL / drug effects
  • Lipoproteins, LDL / immunology
  • Malondialdehyde / pharmacology
  • Mice
  • Mice, Knockout
  • Models, Biological
  • Receptors, LDL / deficiency
  • Th2 Cells / immunology

Substances

  • Apolipoproteins E
  • Interleukin-5
  • Lipoproteins, LDL
  • Receptors, LDL
  • oxidized low density lipoprotein
  • Malondialdehyde