Interleukin 6/B cell stimulatory factor-II is expressed and released by normal and transformed human bronchial epithelial cells

Biochem Biophys Res Commun. 1992 Sep 16;187(2):596-602. doi: 10.1016/0006-291x(92)91236-j.

Abstract

Airway epithelial cells have a potential to participate in local immune and inflammatory responses via releasing biologically active compounds. We studied the expression and release of interleukin 6 (IL-6), a multifunctional cytokine possibly involved in tissue immune responses. Primary culture of normal human bronchial epithelial cells and its transformed cell line BEAS-2B released significant amount of biologically and immunologically intact IL-6 into media. A protein synthesis inhibitor cycloheximide abolished the IL-6 release, suggesting a de novo synthesis. Northern blot analysis demonstrated the expression of the specific IL-6 mRNA. Human bronchial epithelial cells can produce IL-6 and contribute to the local activity of IL-6, suggesting that these cells may play a role in the regulation of airway immune responses.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antibodies, Monoclonal
  • Blotting, Northern
  • Bronchi / drug effects
  • Bronchi / metabolism*
  • Cell Line, Transformed
  • Cells, Cultured
  • Cycloheximide / pharmacology
  • Enzyme-Linked Immunosorbent Assay
  • Epithelium / drug effects
  • Epithelium / metabolism
  • Fetal Blood
  • Gene Expression*
  • Humans
  • Interleukin-6 / genetics*
  • Interleukin-6 / immunology
  • Interleukin-6 / metabolism
  • RNA, Messenger / metabolism
  • Transforming Growth Factor beta / pharmacology
  • Tumor Necrosis Factor-alpha / pharmacology

Substances

  • Antibodies, Monoclonal
  • Interleukin-6
  • RNA, Messenger
  • Transforming Growth Factor beta
  • Tumor Necrosis Factor-alpha
  • Cycloheximide