Homocysteine increases methionine synthase mRNA level in Caco-2 cells

Cell Physiol Biochem. 2004;14(4-6):407-14. doi: 10.1159/000080350.

Abstract

Background: Methionine synthase (MTR) synthesizes methionine from homocysteine, using cobalamin as a cofactor and 5-methyltetrahydrofolate as a cosubstrate.

Aim: To determine the influence of homocystine (Hcy, dimer of homocysteine) in the presence of either cobalamin or methionine on the transcription and the activity of methionine synthase in Caco-2, a human adenocarcinoma cell line.

Methods: Methionine synthase activity and quantification of its mRNA by real-time RT-PCR were determined in cells cultivated under four differents conditions: Hcy with cobalamin (Hcy+ Cbl+), Hcy with methionine (Hcy+Met+), methionine with Cbl (Met+ Cbl+) and methionine only (Met+).

Results: Activity (nmol/h/mg protein) was maximal in cells cultivated in Hcy+Cbl+ (2.45 +/- 0.35), compared to cells cultivated in Hcy+Met+ (0.18 +/- 0.01, p<0.001), in Met+ Cbl+ (1.60 +/- 0.06, p<0.05), and in Met+ (0.40 +/- 0.05, p<0.001), suggesting an adaptation of the cells to requirement in synthesized methionine. The mRNA level of MTR in Hcy+ Cbl+ and Hcy+Met+ (2.82 +/-0.49 and 3.33 +/- 0.48 AU, respectively ) was about 2.5 / 3.0-fold higher than that in Met+ Cbl+ and in Met+ (1.00 +/-0.13 and 1.20 +/-0.20 AU, respectively, p<0.001).

Conclusion: Methionine synthase expression of Caco-2 cell is under a transcriptional control influenced by Hcy.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / analysis
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / biosynthesis
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase / genetics*
  • Adenocarcinoma / chemistry
  • Adenocarcinoma / enzymology
  • Adenocarcinoma / genetics*
  • Caco-2 Cells
  • Gene Expression Regulation, Neoplastic*
  • Homocysteine / pharmacology
  • Homocysteine / physiology*
  • Humans
  • Methionine / pharmacology
  • Methionine / physiology
  • RNA, Messenger / analysis
  • RNA, Messenger / metabolism
  • Transcription, Genetic / genetics
  • Up-Regulation*
  • Vitamin B 12 / pharmacology
  • Vitamin B 12 / physiology

Substances

  • RNA, Messenger
  • Homocysteine
  • Methionine
  • 5-Methyltetrahydrofolate-Homocysteine S-Methyltransferase
  • Vitamin B 12