Neuregulin-1 is neuroprotective and attenuates inflammatory responses induced by ischemic stroke

Biochem Biophys Res Commun. 2004 Sep 17;322(2):440-6. doi: 10.1016/j.bbrc.2004.07.149.

Abstract

Recent work from our laboratory demonstrated that the expression neuregulin-1 in neurons was induced in the ischemic penumbra by focal stroke in the rat. Here, we show that a single intravascular injection of neuregulin-1beta (approximately 2.5 ng/kg) reduced cortical infarct volume by >98% when given immediately before middle cereral artery occlusion. Subcortical infarct volume was reduced by approximately 40%. Analysis of DNA fragmentation in brain tissues indicated that neuregulin-1 blocked apoptosis in cortical neurons in the penumbra. Neuregulin-1 prevented macrophage/microglial infiltration and astrocytic activation following focal ischemia. The neuroprotective effect of neuregulin-1 was also associated with a suppression of interleukin-1beta mRNA levels. These data suggest that neuregulin-1 protects neurons from delayed, ischemia-induced apoptotic cell death in the cortex by inhibiting pro-inflammatory responses. Neuregulins represent a novel, potent neuroprotective strategy that has potential therapeutic value in treating individuals after acute ischemic stroke.

Publication types

  • Research Support, U.S. Gov't, Non-P.H.S.
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Apoptosis / physiology
  • Brain / metabolism
  • Brain / pathology
  • Inflammation / metabolism*
  • Inflammation / pathology
  • Interleukin-1 / biosynthesis
  • Interleukin-1 / genetics
  • Male
  • Neuregulin-1 / metabolism*
  • Neuroglia / metabolism
  • RNA, Messenger / metabolism
  • Rats
  • Stroke / metabolism*
  • Stroke / pathology

Substances

  • Interleukin-1
  • Neuregulin-1
  • RNA, Messenger