Phagocytic cells of the thymic reticulum interact with thymocytes via extracellular matrix ligands and receptors

Cell Immunol. 2004 May;229(1):21-30. doi: 10.1016/j.cellimm.2004.06.002.

Abstract

We previously showed that, in the context of thymic epithelial cells, thymocyte migration is partially controlled by extracellular matrix (ECM)-mediated interactions. Herein we evaluated whether these interactions could be involved in cell migration related events in the context of non-epithelial cells of the thymic microenvironment, the phagocytic cells of the thymic reticulum (PTR). We first showed, by immunocytochemistry, cytofluorometry, and RT-PCR, that PTR produce ECM components, including fibronectin and laminin, and express the corresponding integrin-type receptors, VLA-4, VLA-5, and VLA-6. Thymocytes adhere onto PTR monolayers, with immature CD4(+)CD8(+) cells being predominant. Importantly, such an adhesion is partially mediated by ECM ligands and receptors, since it was impaired by anti-ECM or anti-ECM receptor antibodies. Conjointly, our data reveal that the ECM-dependence for thymocyte adhesion onto the thymic microenvironment is not restricted to the epithelial cells, being also seen when they encounter non-epithelial phagocytic cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Adhesion / physiology
  • Cell Communication / physiology*
  • Cell Movement / physiology
  • Extracellular Matrix / physiology*
  • Female
  • Ligands
  • Mice
  • Mice, Inbred BALB C
  • Phagocytes / physiology*
  • Thymus Gland / physiology*

Substances

  • Ligands