Colorectal cancer and inherited mutations in base-excision repair

Lancet Oncol. 2004 Oct;5(10):600-6. doi: 10.1016/S1470-2045(04)01595-5.

Abstract

Polyposis associated with mutations in the gene MUTYH is an autosomal recessive syndrome characterised by the development of multiple colorectal adenomas and cancer. It is the first cancer-predisposition disorder to be associated with defects in the pathway of base-excision repair. We review our knowledge to date of the disease, discuss base-excision repair in relation to cellular defence against oxidative damage, and give an overview of the molecular genetics and clinicopathological features of tumours associated with MUTYH mutations. No longer a research finding, genetic testing for MUTYH is now a necessary part of molecular diagnosis in familial cancer clinics throughout Australia and the UK. Current recommendations for the screening and management of the disease are also discussed.

Publication types

  • Review

MeSH terms

  • Adenoma / diagnosis
  • Adenoma / genetics*
  • Adenoma / therapy
  • Adenomatous Polyposis Coli / diagnosis
  • Adenomatous Polyposis Coli / genetics
  • Colorectal Neoplasms / diagnosis
  • Colorectal Neoplasms / genetics*
  • Colorectal Neoplasms / therapy
  • DNA Glycosylases / genetics*
  • DNA Repair / genetics*
  • Diagnosis, Differential
  • Genes, Recessive / genetics*
  • Humans
  • Mutation, Missense / genetics*
  • Oxidative Stress / physiology

Substances

  • DNA Glycosylases
  • mutY adenine glycosylase