Combining a targeted radiotherapy and gene therapy approach for adenocarcinoma of prostate

Prostate Cancer Prostatic Dis. 2004;7(4):355-63. doi: 10.1038/sj.pcan.4500760.

Abstract

A targeted radiotherapy/gene therapy approach for prostate cancer, using the radiopharmaceutical [(131)I]meta-iodobenzylguanidine ([(131)I]MIBG), would restrict the effects of radiotherapy to malignant cells, thereby increasing efficacy and decreasing morbidity of radiotherapy. Prostate cancer cells were transfected with a transgene encoding the noradrenaline transporter (NAT) under the control of tumour-specific telomerase promoters, enabling them to actively take up [(131)I]MIBG. This led to tumour-specific cell kill. This strategy has the advantage of generating a radiological bystander effect, leading to the destruction of neighbouring tumour cells that have escaped transfection. This targeted approach could be a promising tumour-specific treatment option for prostate cancer.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 3-Iodobenzylguanidine / therapeutic use*
  • Adenocarcinoma / genetics
  • Adenocarcinoma / pathology
  • Adenocarcinoma / therapy*
  • Bystander Effect
  • Combined Modality Therapy
  • Genetic Therapy*
  • Humans
  • Iodine Radioisotopes
  • Male
  • Norepinephrine Plasma Membrane Transport Proteins
  • Plasmids
  • Promoter Regions, Genetic
  • Prostatic Neoplasms / genetics
  • Prostatic Neoplasms / pathology
  • Prostatic Neoplasms / therapy*
  • Radiopharmaceuticals / therapeutic use*
  • Symporters / therapeutic use*
  • Telomerase / genetics
  • Transfection
  • Tumor Cells, Cultured
  • Tumor Stem Cell Assay

Substances

  • Iodine Radioisotopes
  • Norepinephrine Plasma Membrane Transport Proteins
  • Radiopharmaceuticals
  • SLC6A2 protein, human
  • Symporters
  • 3-Iodobenzylguanidine
  • Telomerase