Late treatment with a protective antigen-directed monoclonal antibody improves hemodynamic function and survival in a lethal toxin-infused rat model of anthrax sepsis

J Infect Dis. 2005 Feb 1;191(3):422-34. doi: 10.1086/427189. Epub 2004 Dec 22.

Abstract

Background: In animal models, treatment with 5H3, a fully human protective antigen-directed monoclonal antibody (PA-MAb), improved survival when administered close to the time of Bacillus anthracis lethal toxin (LeTx) bolus or live bacterial challenge. However, treatment with PA-MAb would be most valuable clinically if it were beneficial even when administered after the onset of shock and lethality due to LeTx.

Methods: We investigated the effects of PA-MAb versus placebo administered in rats (n=324) at the time of or 3, 6, 9, or 12 h after the initiation of a 24-h LeTx infusion.

Results: In rats receiving placebo, mean arterial blood pressure (MBP) and heart rate (HR) were decreased in nonsurvivors, compared with those in survivors, at 6 h and then worsened further, with lethality first evident at 8 h (median, 16 h; range, 8-152 h). At each treatment time, survival rates were greater for PA-MAb than for placebo, although improvement was decreased at later treatment times (P=.001, for the effect of time). Compared with placebo, PA-MAb significantly increased MBP during the 12 h after the initiation of treatment, but the increase was greatest for treatment at 3 h; similarly, PA-MAb significantly increased HR at all treatment times.

Conclusion: In this rat model, improvements in outcome due to PA-MAb were significant when it was administered up to 6 h (and approached significance when administered up to 12 h) after initial exposure to LeTx. Clinically, PA-MAb may be beneficial even when administered after the onset of shock and lethality due to LeTx.

MeSH terms

  • Animals
  • Anthrax / mortality
  • Anthrax / therapy*
  • Antibodies, Bacterial / administration & dosage*
  • Antibodies, Bacterial / immunology
  • Antibodies, Monoclonal / administration & dosage*
  • Antibodies, Monoclonal / immunology
  • Antigens, Bacterial / administration & dosage
  • Antigens, Bacterial / immunology
  • Antigens, Bacterial / toxicity*
  • Bacillus anthracis / metabolism
  • Bacillus anthracis / pathogenicity
  • Bacterial Toxins / administration & dosage
  • Bacterial Toxins / immunology
  • Bacterial Toxins / toxicity*
  • Blood Pressure
  • Heart Rate
  • Hemodynamics / drug effects*
  • Infusions, Intravenous
  • Rats
  • Rats, Sprague-Dawley
  • Sepsis / mortality
  • Sepsis / therapy*
  • Time Factors

Substances

  • Antibodies, Bacterial
  • Antibodies, Monoclonal
  • Antigens, Bacterial
  • Bacterial Toxins
  • anthrax toxin