Endogenous bone-marrow-derived stem cells contribute only a small proportion of regenerated myocardium in the acute infarction model

J Heart Lung Transplant. 2005 Jan;24(1):67-72. doi: 10.1016/j.healun.2003.09.032.

Abstract

Background: Our recent study showed that granulocyte-colony stimulating factor (G-CSF) promoted bone-marrow cells (BMC) to migrate into the infarcted heart and that they differentiated into cardiomyocytes. However, we still do not know to what degree bone-marrow-derived cardiomyocytes contribute to myocardial regeneration after injury. In this study, we verified the proportional contribution of cells from bone marrow (BM) and from non-bone marrow (n-BM) in regenerating neomyocardium after myocardial infarction.

Methods: Eight C57BL/6 mice were irradiated (900 cGy), and green fluorescent protein (GFP) mouse-derived BMCs (GFP-BMC, 1 x 10(6) cells) were injected. Four weeks later, the left descending coronary artery was ligated. Recombinant human G-CSF (200 microg/kg/day, 8 days) was injected. At 4 weeks after ligation, hearts were fixed for histology. We calculated the proportions of cardiomyocytes derived from BM and n-BM after taking the chimeric rate into consideration.

Results: The chimeric rate was 54.6% +/- 5.9%. At the infarcted border area, the total cell number was 1000.3 +/- 56.5/mm(2), and mobilized BM-derived GFP-BMC was 103.3 +/- 13.1/mm(2). After compensation with the chimeric rate, we found BM-derived troponin I-positive cells at 23.9 +/- 4.1/mm(2), nestin-positive cells at 12.9 +/- 2.6/mm(2), and Ki67-positive cells at 18.3 +/- 2.6/mm(2), respectively. We found significant differences in the contribution of troponin I-(6.7% +/- 1.7% vs 93.3% +/- 1.7%), nestin- (2.4 +/- 0.5 vs 97.6 +/- 0.5), and Ki67-positive (3.9 +/- 1.0 vs 96.1 +/- 1.0) cells derived from BM and n-BM.

Conclusions: Bone marrow was one of the origins of regenerated cardiomyocytes; however, the contribution of cells from BM was very small compared with those of n-BM origin in the infarction model.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Bone Marrow Cells / physiology*
  • Cell Movement / physiology*
  • Disease Models, Animal
  • Green Fluorescent Proteins / metabolism
  • Green Fluorescent Proteins / pharmacology
  • Intermediate Filament Proteins / metabolism
  • Luminescent Agents / metabolism
  • Luminescent Agents / pharmacology
  • Mice
  • Mice, Inbred C57BL
  • Models, Cardiovascular
  • Myocardial Infarction / metabolism
  • Myocardial Infarction / physiopathology*
  • Myocardium / cytology*
  • Myocardium / metabolism
  • Myocytes, Cardiac / physiology*
  • Nerve Tissue Proteins / metabolism
  • Nestin
  • Regeneration*
  • Troponin I / metabolism

Substances

  • Intermediate Filament Proteins
  • Luminescent Agents
  • NES protein, human
  • Nerve Tissue Proteins
  • Nes protein, mouse
  • Nestin
  • Troponin I
  • Green Fluorescent Proteins