DBC2 significantly influences cell-cycle, apoptosis, cytoskeleton and membrane-trafficking pathways

J Mol Biol. 2005 Feb 11;346(1):83-9. doi: 10.1016/j.jmb.2004.11.043. Epub 2004 Dec 8.

Abstract

The tumor suppressor DBC2 belongs to a previously uncharacterized gene family, RHOBTB (Bric-a-brac, Tramtrack, Broad-complex). The biological roles of RHOBTB proteins, including DBC2, remain unclear. To understand the physiological functions of DBC2, a global approach was applied. Expression of DBC2 was manipulated in HeLa cells and RNA profiling of the cells was performed by microarray analyses. DBC2 was introduced into HeLa cells by a mammalian expression vector with a constitutive promoter. DBC2 knockdown was achieved by RNA interference with small interfering RNA. RNA profiles of these samples were performed by microarray analysis using Affymetrix GeneChip HG-U133A 2.0. The microarray data were analyzed by Microarray Suite 5.0 (MAS 5.0) and Robust Multichip Average (RMA). A list of genes whose expression was significantly altered (p<0.001) was generated and overlaid onto a cellular pathway map in the Ingenuity Systems' Pathway Knowledge Base (Winter'04 Release). Two networks were found to react substantially to DBC2 expression; namely, more than half of participating genes are affected. One of the networks regulates cell growth through cell-cycle control and apoptosis. The other network is related to cytoskeleton and membrane trafficking. Our findings suggest that the biological roles of DBC2 are related directly and/or indirectly to these cellular machineries.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adaptor Proteins, Signal Transducing
  • Apoptosis*
  • Carrier Proteins / metabolism
  • Cell Cycle Proteins
  • Cell Cycle*
  • Cell Membrane / metabolism*
  • Cytoskeletal Proteins
  • Cytoskeleton / metabolism*
  • GTP-Binding Proteins / deficiency
  • GTP-Binding Proteins / genetics
  • GTP-Binding Proteins / metabolism*
  • HeLa Cells
  • Humans
  • Oligonucleotide Array Sequence Analysis
  • Protein Transport
  • RNA Interference
  • Ribonucleases / metabolism
  • Signal Transduction
  • Tumor Suppressor Proteins / deficiency
  • Tumor Suppressor Proteins / genetics
  • Tumor Suppressor Proteins / metabolism*

Substances

  • Adaptor Proteins, Signal Transducing
  • Carrier Proteins
  • Cell Cycle Proteins
  • Cytoskeletal Proteins
  • RHOBTB2 protein, human
  • Tumor Suppressor Proteins
  • USH1C protein, human
  • Ribonucleases
  • GTP-Binding Proteins