The dual 5-alpha-reductase inhibitor dutasteride induces atrophic changes and decreases relative cancer volume in human prostate

Urology. 2005 Jan;65(1):76-82. doi: 10.1016/j.urology.2004.08.042.

Abstract

Objectives: To perform the first evaluation of the effects of the 5-alpha-reductase inhibitor class of drugs on cancer histopathologic features at radical prostatectomy in a placebo-controlled multicenter trial.

Methods: We analyzed prostatectomy slides in a blinded manner from 17 men treated with dutasteride, an inhibitor of types 1 and 2 isoenzymes of 5-alpha-reductase, and 18 men treated with placebo for 5 to 11 weeks before radical prostatectomy. The histopathologic features of benign epithelium, high-grade prostatic intraepithelial neoplasia, and cancer were recorded, and the treatment effect was scored. Digital imaging analysis was used to measure the stroma/epithelium ratio and epithelial height, as well as the nuclear area in cancer.

Results: In benign epithelium, treatment caused distinctive cytoarchitectural changes of atrophy and a decrease in the epithelial height (P = 0.053). The peripheral zone showed the most marked response to treatment. In cancer tissue, the tumor volume was significantly lower in the dutasteride-treated men than in the placebo-treated men (mean 15% versus 24%, respectively, P = 0.025), the percentage of atrophic epithelium was increased (P = 0.041), and the stroma/gland ratio was doubled (P = 0.046). The treatment alteration effect score was doubled (P = 0.055) and did not correlate with any Gleason score changes.

Conclusions: After short-term dutasteride treatment, benign epithelium showed involution and epithelial shrinkage, and prostate cancer tissue demonstrated a decrease in epithelium relative to stroma. These findings indicate that dutasteride induces significant phenotypic alterations in both the benign and the neoplastic prostate, supportive of a chemopreventive or chemoactive role.

Publication types

  • Clinical Trial
  • Comparative Study
  • Multicenter Study
  • Randomized Controlled Trial

MeSH terms

  • 5-alpha Reductase Inhibitors*
  • Adenocarcinoma / drug therapy*
  • Adenocarcinoma / enzymology
  • Adenocarcinoma / pathology
  • Adenocarcinoma / surgery
  • Aged
  • Antineoplastic Agents, Hormonal / administration & dosage
  • Antineoplastic Agents, Hormonal / pharmacology
  • Antineoplastic Agents, Hormonal / therapeutic use*
  • Atrophy
  • Azasteroids / administration & dosage
  • Azasteroids / pharmacology
  • Azasteroids / therapeutic use*
  • Combined Modality Therapy
  • Double-Blind Method
  • Dutasteride
  • Epithelial Cells / drug effects
  • Epithelial Cells / pathology
  • Humans
  • Image Processing, Computer-Assisted
  • Male
  • Middle Aged
  • Neoadjuvant Therapy
  • Neoplasm Proteins / antagonists & inhibitors*
  • Pilot Projects
  • Prostate / drug effects*
  • Prostate / pathology
  • Prostatectomy
  • Prostatic Intraepithelial Neoplasia / drug therapy
  • Prostatic Intraepithelial Neoplasia / enzymology
  • Prostatic Intraepithelial Neoplasia / pathology
  • Prostatic Intraepithelial Neoplasia / surgery
  • Prostatic Neoplasms / drug therapy*
  • Prostatic Neoplasms / enzymology
  • Prostatic Neoplasms / pathology
  • Prostatic Neoplasms / surgery
  • Stromal Cells / drug effects
  • Stromal Cells / pathology

Substances

  • 5-alpha Reductase Inhibitors
  • Antineoplastic Agents, Hormonal
  • Azasteroids
  • Neoplasm Proteins
  • Dutasteride