Up-regulation of peripherin is associated with alterations in synaptic plasticity in CA1 and CA3 regions of hippocampus

Neurobiol Dis. 2005 Mar;18(2):409-20. doi: 10.1016/j.nbd.2004.10.009.

Abstract

Peripherin is a type III intermediate filament protein normally undetectable in most brain neurons. Here, we report a similar pattern of peripherin expression in the brains of both mice treated with systemic injections of kainic acid (KA) and in peripherin transgenic mice (Per mice) over-expressing the normal peripherin gene under its own promoter. Double-immunofluorescence labeling revealed a partial co-localization of peripherin with the microtubule-associated protein MAP2, but not with neurofilament proteins. Electrophysiological studies revealed that synaptic plasticity was markedly altered in Per mice: in CA1, long-term potentiation (LTP) was decreased in Per slices (+29 +/- 2.0%, vs. +58 +/- 5.4%, in WT); while in CA3, LTP was increased in Per (+63 +/- 3.5% vs. +43 +/- 2.4.0%). In the hippocampus of Per mice, the levels of MAP2 were decreased, though synaptophysin and PSD95 remained unchanged. These intriguing findings suggest a role of peripherin in the alteration of hippocampal synaptic plasticity.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Animals, Genetically Modified
  • Behavior, Animal / physiology
  • Disease Models, Animal
  • Down-Regulation / drug effects
  • Down-Regulation / genetics
  • Epilepsy / chemically induced
  • Epilepsy / metabolism*
  • Epilepsy / physiopathology
  • Fluorescent Antibody Technique
  • Gene Expression / drug effects
  • Gene Expression / physiology
  • Genetic Predisposition to Disease
  • Hippocampus / drug effects
  • Hippocampus / metabolism*
  • Hippocampus / physiopathology
  • Intermediate Filament Proteins / genetics
  • Intermediate Filament Proteins / metabolism*
  • Kainic Acid
  • Long-Term Potentiation / genetics
  • Membrane Glycoproteins / genetics
  • Membrane Glycoproteins / metabolism*
  • Mice
  • Microtubule-Associated Proteins / metabolism
  • Nerve Tissue Proteins / genetics
  • Nerve Tissue Proteins / metabolism*
  • Neuronal Plasticity / genetics*
  • Organ Culture Techniques
  • Peripherins
  • Promoter Regions, Genetic / drug effects
  • Promoter Regions, Genetic / genetics
  • Synaptic Transmission / genetics*
  • Synaptophysin / metabolism
  • Thalamus / metabolism
  • Thalamus / physiopathology
  • Up-Regulation / drug effects
  • Up-Regulation / genetics

Substances

  • Intermediate Filament Proteins
  • Membrane Glycoproteins
  • Microtubule-Associated Proteins
  • Mtap2 protein, mouse
  • Nerve Tissue Proteins
  • Peripherins
  • Synaptophysin
  • Kainic Acid