Heterologous T cell immunity in severe hepatitis C virus infection

J Exp Med. 2005 Mar 7;201(5):675-80. doi: 10.1084/jem.20041058.

Abstract

Hepatitis C virus (HCV) can cause liver disease of variable severity. Expansion of preexisting memory CD8 T cells by cross-reactivity with a new heterologous virus infection has been shown in mice to shape the repertoire of the primary response and to influence virus-related immunopathology. To determine whether this mechanism can influence the course of HCV infection, we analyzed the features of the HCV-specific CD8 T cell response in eight patients with acute HCV infection, two of whom had a particularly severe illness. Patients with severe hepatitis, but not those with mild disease, showed an extremely vigorous CD8 T cell response narrowly focused on a single epitope (NS3 1073-1081), which cross-reacted with an influenza neuraminidase sequence. Our results suggest that CD8 T cell cross-reactivity influences the severity of the HCV-associated liver pathology and depicts a model of disease induction that may apply to different viral infections.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Disease
  • CD8-Positive T-Lymphocytes / immunology*
  • CD8-Positive T-Lymphocytes / metabolism
  • Cross Reactions / immunology
  • Female
  • Hepacivirus / immunology*
  • Hepatitis C / immunology*
  • Hepatitis C / metabolism
  • Hepatitis C / pathology
  • Hepatitis C / virology*
  • Humans
  • Interferon-gamma / metabolism
  • Kinetics
  • Male

Substances

  • Interferon-gamma