Quantitative expression levels of regulated on activation, normal T cell expressed and secreted and eotaxin transcripts in toluene diisocyanate-induced allergic rats

Acta Otolaryngol. 2005 Apr;125(4):370-7. doi: 10.1080/00016480510003156.

Abstract

Conclusion: These results suggest that eotaxin may play a predominant role in controlling antigen-induced eosinophil recruitment into tissue. Objective To investigate the expression levels of regulated on activation, normal T cell expressed and secreted (RANTES) mRNA and eotaxin mRNA in the nasal mucosa of toluene diisocyanate (TDI)-induced allergic rats and to evaluate which of them is primarily related to selective eosinophilic infiltration by comparing their expression levels with the numbers of infiltrated eosinophils and vascular cell adhesion molecule-1 (VCAM-1).

Material and methods: We quantified the expression levels of two strong eosinophilic CC chemokines (RANTES and eotaxin) and VCAM-1 at mRNA levels in the nasal mucosa of TDI-induced allergic rats using competitive polymerase chain reaction and compared their expression levels with the number of infiltrated eosinophils.

Results: The number of infiltrated eosinophils was significantly increased between 3 h and Day 4 in TDI-induced allergic rats, but had decreased by Day 5. VCAM-1 mRNA expression was also increased between 3 h and Day 4. The number of infiltrated eosinophils correlated with the expression levels of VCAM-1 mRNA (p < 0.01). In contrast, expression of RANTES mRNA and eotaxin mRNA was increased between 3 h and Day 2, peaked between Days 1 and 2 and then declined. Although the expression of both chemokines correlated with the numbers of infiltrated eosinophils (p < 0.01), peak expression levels of eotaxin mRNA were 14-fold higher than baseline levels whereas RANTES mRNA expression increased 3-fold.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Asthma / chemically induced
  • Asthma / genetics
  • Asthma / immunology*
  • Chemokine CCL11
  • Chemokine CCL5 / genetics
  • Chemokines, CC / genetics*
  • Eosinophilia / chemically induced
  • Eosinophilia / genetics
  • Eosinophilia / immunology
  • Gene Expression Regulation / drug effects
  • Gene Expression Regulation / immunology
  • Leukocyte Count
  • Lymphocyte Activation / drug effects
  • Lymphocyte Activation / genetics
  • Lymphocyte Activation / immunology*
  • Male
  • Nasal Mucosa / drug effects
  • Nasal Mucosa / immunology
  • RNA, Messenger / genetics
  • Rats
  • Rats, Sprague-Dawley
  • Respiratory Hypersensitivity / chemically induced
  • Respiratory Hypersensitivity / genetics
  • Respiratory Hypersensitivity / immunology*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Rhinitis, Allergic, Perennial / chemically induced
  • Rhinitis, Allergic, Perennial / genetics
  • Rhinitis, Allergic, Perennial / immunology*
  • T-Lymphocytes / drug effects
  • T-Lymphocytes / immunology*
  • Toluene 2,4-Diisocyanate / immunology*
  • Toluene 2,4-Diisocyanate / toxicity
  • Vascular Cell Adhesion Molecule-1 / genetics
  • beta 2-Microglobulin / genetics

Substances

  • Ccl11 protein, rat
  • Chemokine CCL11
  • Chemokine CCL5
  • Chemokines, CC
  • RNA, Messenger
  • Vascular Cell Adhesion Molecule-1
  • beta 2-Microglobulin
  • Toluene 2,4-Diisocyanate