Induction of lytic Epstein-Barr virus (EBV) infection by synergistic action of rituximab and dexamethasone renders EBV-positive lymphoma cells more susceptible to ganciclovir cytotoxicity in vitro and in vivo

J Virol. 2005 May;79(9):5875-9. doi: 10.1128/JVI.79.9.5875-5879.2005.

Abstract

The purposeful induction of the lytic form of Epstein-Barr virus (EBV) infection combined with ganciclovir (GCV) treatment has been advocated as a novel strategy for EBV-positive B-cell lymphoma. We demonstrated that rituximab had a synergistic effect with dexamethasone on induction of the lytic EBV infection in CD20-positive lymphoma cells. Addition of GCV to the dexamethasone/rituximab-treated cells was more effective than dexamethasone/rituximab alone in killing EBV-positive lymphoma cells in vitro and in lymphoma-bearing nude mice but not in EBV-negative cells. These data suggest that induction of the lytic EBV infection with dexamethasone/rituximab in combination with GCV could be a potential virally targeted therapy for EBV-associated B-cell lymphoma.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Antibodies, Monoclonal / pharmacology*
  • Antibodies, Monoclonal, Murine-Derived
  • Antineoplastic Agents, Hormonal / pharmacology
  • Antiviral Agents / pharmacology*
  • Dexamethasone / pharmacology*
  • Drug Synergism
  • Epstein-Barr Virus Infections / virology*
  • Ganciclovir / pharmacology*
  • Herpesvirus 4, Human / physiology*
  • Humans
  • Lymphoma, B-Cell / drug therapy
  • Lymphoma, B-Cell / virology*
  • Rituximab
  • Virus Activation

Substances

  • Antibodies, Monoclonal
  • Antibodies, Monoclonal, Murine-Derived
  • Antineoplastic Agents, Hormonal
  • Antiviral Agents
  • Rituximab
  • Dexamethasone
  • Ganciclovir