Interfering with hepatitis C virus IRES activity using RNA molecules identified by a novel in vitro selection method

Biol Chem. 2005 Feb;386(2):183-90. doi: 10.1515/BC.2005.023.

Abstract

Hepatitis C virus (HCV) infection is one of the world's major health problems, and the identification of efficient HCV inhibitors is a major goal. Here we report the isolation of efficient anti-HCV internal ribosome entry site (IRES) RNA molecules identified by a new in vitro selection method. The newly developed procedure consists of two sequential steps that use distinct criteria for selection: selection for binding and selection for cleaving. The selection protocol was applied to a population of more than 10(15) variants of an anti-hepatitis C virus ribozyme covalently linked to an aptamer motif. The ribozyme was directed against positions 357 to 369 of the HCV IRES, and the cleavage substrate was a 691-nucleotide-long RNA fragment that comprises the entire HCV IRES domain. After six selection cycles, seven groups of RNA variants were identified. A representative of each group was tested for its capacity to inhibit IRES activity using in vitro translation assays. All selected RNAs promoted significant inhibition, some by as much as 95%.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • 5' Untranslated Regions*
  • Base Sequence
  • Databases, Nucleic Acid
  • Hepacivirus / drug effects
  • Hepacivirus / genetics*
  • Hepacivirus / metabolism
  • Molecular Sequence Data
  • Nucleic Acid Amplification Techniques / methods
  • Nucleic Acid Conformation
  • Oligoribonucleotides / isolation & purification
  • Oligoribonucleotides / pharmacology
  • Protein Biosynthesis
  • RNA, Catalytic / isolation & purification
  • RNA, Catalytic / metabolism*
  • RNA, Viral / isolation & purification
  • RNA, Viral / metabolism*
  • Ribosomes / metabolism*

Substances

  • 5' Untranslated Regions
  • Oligoribonucleotides
  • RNA, Catalytic
  • RNA, Viral