Erythropoietin decreases cytotoxicity and nitric oxide formation induced by inflammatory stimuli in rat oligodendrocytes

Physiol Res. 2006;55(1):33-38. doi: 10.33549/physiolres.930749. Epub 2005 Apr 26.

Abstract

In the present study, we investigated whether erythropoietin (Epo) has a protective effect against cytotoxicity induced by interferon-gamma (IFN-gamma ) and lipopolysaccharide (LPS) in primary rat oligodendrocyte cultures. The possible modulatory effect of erythropoietin on inducible nitric oxide synthase (iNOS) mRNA expression and nitrite production were also analyzed. Erythropoietin exerted a significant protective effect against IFN-gamma and LPS-induced oligodendrocyte injury as determined by lactate dehydrogenase assay. Treatment with erythropoietin inhibited the expression of iNOS mRNA and nitrite production resulting from proinflammatory stimulation by IFN-gamma and LPS. These results suggest that erythropoietin has protective effects against inflammatory oligodendrocyte injury in vitro and may play a protective role in neurological disorders characterized by oligodendrocyte death, such as multiple sclerosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Animals, Newborn
  • Brain
  • Cell Survival
  • Cells, Cultured
  • Erythropoietin / pharmacology*
  • Gene Expression Regulation
  • Interferon-gamma
  • Lipopolysaccharides
  • Neuroprotective Agents / pharmacology*
  • Nitric Oxide / biosynthesis
  • Nitric Oxide / metabolism*
  • Nitric Oxide Synthase Type II / biosynthesis
  • Nitric Oxide Synthase Type II / metabolism
  • Oligodendroglia / drug effects*
  • Oligodendroglia / metabolism
  • RNA, Messenger / metabolism
  • Rats
  • Rats, Wistar

Substances

  • Lipopolysaccharides
  • Neuroprotective Agents
  • RNA, Messenger
  • lipopolysaccharide, E. coli O26-B6
  • Erythropoietin
  • Nitric Oxide
  • Interferon-gamma
  • Nitric Oxide Synthase Type II
  • Nos2 protein, rat