Chemokine up-regulation in SARS-coronavirus-infected, monocyte-derived human dendritic cells

Blood. 2005 Oct 1;106(7):2366-74. doi: 10.1182/blood-2004-10-4166. Epub 2005 Apr 28.

Abstract

Lymphopenia and increasing viral load in the first 10 days of severe acute respiratory syndrome (SARS) suggested immune evasion by SARS-coronavirus (CoV). In this study, we focused on dendritic cells (DCs) which play important roles in linking the innate and adaptive immunity. SARS-CoV was shown to infect both immature and mature human monocyte-derived DCs by electron microscopy and immunofluorescence. The detection of negative strands of SARS-CoV RNA in DCs suggested viral replication. However, no increase in viral RNA was observed. Using cytopathic assays, no increase in virus titer was detected in infected DCs and cell-culture supernatant, confirming that virus replication was incomplete. No induction of apoptosis or maturation was detected in SARS-CoV-infected DCs. The SARS-CoV-infected DCs showed low expression of antiviral cytokines (interferon alpha [IFN-alpha], IFN-beta, IFN-gamma, and interleukin 12p40 [IL-12p40]), moderate up-regulation of proinflammatory cytokines (tumor necrosis factor alpha [TNF-alpha] and IL-6) but significant up-regulation of inflammatory chemokines (macrophage inflammatory protein 1alpha [MIP-1alpha], regulated on activation normal T cell expressed and secreted [RANTES]), interferon-inducible protein of 10 kDa [IP-10], and monocyte chemoattractant protein 1 [MCP-1]). The lack of antiviral cytokine response against a background of intense chemokine up-regulation could represent a mechanism of immune evasion by SARS-CoV.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Apoptosis
  • Caspase 3
  • Caspases / biosynthesis
  • Cell Separation
  • Cells, Cultured
  • Chemokine CCL2 / biosynthesis
  • Chemokine CCL3
  • Chemokine CCL4
  • Chemokine CCL5 / biosynthesis
  • Chemokines / biosynthesis*
  • Cytokines / metabolism
  • DNA Primers / chemistry
  • Dendritic Cells / cytology*
  • Enzyme Activation
  • Flow Cytometry
  • Fluorescent Antibody Technique, Indirect
  • Humans
  • Inflammation
  • Interferon-alpha / biosynthesis
  • Interferon-beta / biosynthesis
  • Interferon-gamma / biosynthesis
  • Interleukin-12 / biosynthesis
  • Interleukin-12 / metabolism
  • Interleukin-12 Subunit p40
  • Interleukin-6 / biosynthesis
  • Leukocytes, Mononuclear / cytology
  • Macrophage Inflammatory Proteins / biosynthesis
  • Microscopy, Electron, Transmission
  • Microscopy, Fluorescence
  • Monocytes / cytology*
  • Polymerase Chain Reaction
  • Protein Subunits / biosynthesis
  • RNA / metabolism
  • RNA, Viral / metabolism
  • Reverse Transcriptase Polymerase Chain Reaction
  • Severe acute respiratory syndrome-related coronavirus / genetics
  • Severe acute respiratory syndrome-related coronavirus / metabolism*
  • Time Factors
  • Up-Regulation*

Substances

  • Chemokine CCL2
  • Chemokine CCL3
  • Chemokine CCL4
  • Chemokine CCL5
  • Chemokines
  • Cytokines
  • DNA Primers
  • Interferon-alpha
  • Interleukin-12 Subunit p40
  • Interleukin-6
  • Macrophage Inflammatory Proteins
  • Protein Subunits
  • RNA, Viral
  • Interleukin-12
  • RNA
  • Interferon-beta
  • Interferon-gamma
  • CASP3 protein, human
  • Caspase 3
  • Caspases