7-84 parathyroid hormone fragments are proportionally increased with the severity of uremic hyperparathyroidism

Clin Nephrol. 2005 May;63(5):351-5. doi: 10.5414/cnp63351.

Abstract

Aims: Recent progress in PTH assay has revealed that the intact PTH assay kit in current use does not differentiate between the truncated 7-84 PTH molecule and the 1-84 PTH molecule. In our series, we examined the effectiveness of a new PTH assay as a noninvasive method of evaluating severity of uremic hyperparathyroidism.

Methods and materials: Two hundred and seventy hemodialysis (HD) patients recruited from three HD centers were included and divided into subgroups according to the conventional iPTH assay results. Pre-dialysis blood samples were collected and subjected to two different PTH assays: "intact" PTH assay (iPTH) and "whole" PTH (wPTH) assay. Two biochemical markers of bone remodeling were also examined.

Results: In all cases, PTH levels determined by the wPTH assay were in the average 32.3% lower than those determined by the iPTH assay. The difference of the results of the two PTH assay methods, which indicated the portion of 7-84 PTH fragments of the total PTH molecules measured with the iPTH assay, was gradually increased while the severity of uremic hyperparathyroidism increased. Biochemical markers of bone formation/resorption showed a similar change.

Conclusion: The portion of the 7-84 PTH fragments and markers of increased bone turnover increased in proportion to the severity of uremic hyperparathyroidism. This finding disproves the hypothetical role of 7-84 PTH fragments alone as the noninvasive marker of low-turnover bone disease in HD patients.

Publication types

  • Comparative Study
  • Multicenter Study

MeSH terms

  • Adult
  • Age Factors
  • Aged
  • Aged, 80 and over
  • Analysis of Variance
  • Biomarkers / analysis
  • Biomarkers / metabolism
  • Chronic Kidney Disease-Mineral and Bone Disorder / diagnosis*
  • Chronic Kidney Disease-Mineral and Bone Disorder / etiology
  • Cohort Studies
  • Disease Progression
  • Female
  • Humans
  • Hyperparathyroidism, Secondary / diagnosis*
  • Hyperparathyroidism, Secondary / etiology
  • Kidney Failure, Chronic / complications
  • Kidney Failure, Chronic / diagnosis
  • Kidney Failure, Chronic / therapy
  • Male
  • Middle Aged
  • Parathyroid Hormone / metabolism*
  • Parathyroid Hormone / urine
  • Prognosis
  • Prospective Studies
  • Radioimmunoassay
  • Reference Values
  • Renal Dialysis / adverse effects*
  • Renal Dialysis / methods
  • Risk Assessment
  • Sensitivity and Specificity
  • Severity of Illness Index
  • Sex Factors

Substances

  • Biomarkers
  • Parathyroid Hormone