Topoisomerase IIalpha and APE/ref-1 are associated with pathologic response to primary anthracycline-based chemotherapy for breast cancer

Cancer Lett. 2005 Jun 16;224(1):133-9. doi: 10.1016/j.canlet.2004.11.007. Epub 2004 Dec 13.

Abstract

The aim of this study was to evaluate the role of several biological and histological markers (topoisomerase IIalpha, MIB-1, E2F, apoptotic index, APE/ref-1, p53, Her-2/neu, estrogen and porgesterone receptors, and histological grading) as predictors of pathologic response after anthracycline-based chemotherapy for breast cancer. A series of 50 consecutive breast cancer patients receiving anthracycline-based primary chemotherapy were retrospectively studied. Biological markers were assessed by immunohistochemistry (and by TUNEL assay for apoptotic index) in pre-treatment core biopsies and post-treatment surgical samples. The expression of topoisomerase IIalpha, E2F, MIB-1, estrogen and progesterone receptors decreased, while APE/ref-1 staining increased after treatment. Higher topoisomerase IIalpha (P=0.007) and lower APE/ref-1 (P=0.04) expression were associated with better pathologic response.

MeSH terms

  • Adult
  • Aged
  • Anthracyclines / pharmacology
  • Anthracyclines / therapeutic use*
  • Antigens, Neoplasm / blood*
  • Antineoplastic Agents / pharmacology
  • Antineoplastic Agents / therapeutic use*
  • Biomarkers, Tumor / blood*
  • Breast Neoplasms / drug therapy*
  • Breast Neoplasms / physiopathology*
  • Carcinoma / drug therapy*
  • Carcinoma / physiopathology*
  • DNA Repair
  • DNA Topoisomerases, Type II / blood*
  • DNA-(Apurinic or Apyrimidinic Site) Lyase / blood*
  • DNA-Binding Proteins / blood*
  • Female
  • Humans
  • In Situ Nick-End Labeling
  • Isoenzymes
  • Middle Aged
  • Predictive Value of Tests
  • Treatment Outcome

Substances

  • Anthracyclines
  • Antigens, Neoplasm
  • Antineoplastic Agents
  • Biomarkers, Tumor
  • DNA-Binding Proteins
  • Isoenzymes
  • APEX1 protein, human
  • DNA-(Apurinic or Apyrimidinic Site) Lyase
  • DNA Topoisomerases, Type II