Abstract
The synthetic immunomodulator cytotoxic T lymphocyte antigen 4-Ig (CTLA-4-Ig) initiates effects in human monocyte-derived dendritic cells (DC) that rely on immunosuppressive tryptophan catabolism. However, it is unable to induce suppressive properties in DC matured by CD40 engagement. Thus, CD40-driven events may physiologically set human DC free from restraint by regulatory cells expressing surface CTLA-4.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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Abatacept
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Animals
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CD40 Antigens / physiology*
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Cell Proliferation
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Dendritic Cells / drug effects*
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Dendritic Cells / physiology*
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Female
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Humans
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Immune Tolerance / drug effects*
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Immunoconjugates / pharmacology*
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Interferon-gamma / physiology
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Mice
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Mice, Inbred DBA
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Protein Binding
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Tryptophan / metabolism
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Tryptophan Oxygenase / metabolism
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p38 Mitogen-Activated Protein Kinases / metabolism
Substances
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CD40 Antigens
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Immunoconjugates
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Abatacept
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Interferon-gamma
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Tryptophan
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Tryptophan Oxygenase
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p38 Mitogen-Activated Protein Kinases