Effect of electro-acupuncture on ovarian expression of alpha (1)- and beta (2)-adrenoceptors, and p75 neurotrophin receptors in rats with steroid-induced polycystic ovaries

Reprod Biol Endocrinol. 2005 Jun 7:3:21. doi: 10.1186/1477-7827-3-21.

Abstract

Background: Estradiol valerate (EV)-induced polycystic ovaries (PCO) in rats is associated with an increase in ovarian sympathetic outflow. Low-frequency (2 Hz) electro-acupuncture (EA) has been shown to modulate sympathetic markers as well as ovarian blood flow as a reflex response via the ovarian sympathetic nerves, in rats with EV-induced PCO.

Methods: In the present study, we further tested the hypothesis that repeated 2 Hz EA treatments modulate ovarian sympathetic outflow in rats with PCO, induced by a single i.m. injection of EV, by investigating the mRNA expression, the amount and distribution of proteins of alpha1a-, alpha1b-, alpha1d-, and beta2-adrenoceptors (ARs), as well as the low-affinity neurotrophin receptor (p75NTR).

Results: It was found that EV injection results in significantly higher mRNA expression of ovarian alpha1b- and alpha1d-AR in PCO rats compared to control rats. The p75NTR and beta2-ARs mRNA expression were unchanged in the PCO ovary. Low-frequency EA resulted in a significantly lower expression of beta2-ARs mRNA expression in PCO rats. The p75NTR mRNA was unaffected in both PCO and control rats. PCO ovaries displayed significantly higher amount of protein of alpha1a-, alpha1b- and alpha1d-ARs, and of p75NTR, compared to control rats, that were all counteracted by repeated low-frequency EA treatments, except for alpha1b-AR.

Conclusion: The present study shows that EA normalizes most of the EV-induced changes in ovarian ARs. Furthermore, EA was able to prevent the EV-induced up regulation of p75NTR, probably by normalizing the sympathetic ovarian response to NGF action. Our data indicate a possible role of EA in the regulation of ovarian responsiveness to sympathetic inputs and depict a possible complementary therapeutic approach to overcoming sympathetic-related anovulation in women with PCOS.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Corpus Luteum / cytology
  • Electroacupuncture*
  • Estradiol / analogs & derivatives
  • Female
  • Gene Expression Regulation
  • Ovarian Follicle / cytology
  • Ovary / metabolism*
  • Polycystic Ovary Syndrome / chemically induced
  • Polycystic Ovary Syndrome / metabolism
  • Polycystic Ovary Syndrome / therapy*
  • RNA, Messenger / metabolism
  • Rats
  • Rats, Inbred WKY
  • Receptor, Nerve Growth Factor / metabolism*
  • Receptors, Adrenergic, alpha-1 / metabolism*
  • Receptors, Adrenergic, beta-2 / metabolism*

Substances

  • RNA, Messenger
  • Receptor, Nerve Growth Factor
  • Receptors, Adrenergic, alpha-1
  • Receptors, Adrenergic, beta-2
  • Estradiol