Regulation of hippocampal synapse remodeling by epileptiform activity

Mol Cell Neurosci. 2005 Aug;29(4):494-506. doi: 10.1016/j.mcn.2005.04.007.

Abstract

We examined the regulation of dendritic spines and synapses by epileptiform activity (EA) in rat hippocampal slice cultures. EA, which was induced by a GABA(A) receptor inhibitor, gabazine, reduced pyramidal neuron spine density by approximately 50% after 48 h and also caused an increase in the average length of remaining spines. To directly determine the effects of EA on synapses, we used fluorescent protein-tagged PSD95, which marks postsynaptic densities. EA induced a net loss of synapses on spines but not shafts; conversely, activity blockade (TTX) induced a loss of shaft synapses. Time-lapse confocal imaging in live tissue slices revealed that EA (1) shifts the balance of synapse gain and loss in dendrites leading to a net loss of spine synapses and (2) induces the formation of new filopodia-like dendritic structures having abnormally slow motility. These results identify EA-induced changes in the density and distribution of synaptic structures on dendrites.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Animals, Newborn
  • Dendritic Spines / drug effects
  • Dendritic Spines / pathology
  • Dendritic Spines / physiology*
  • Disks Large Homolog 4 Protein
  • Down-Regulation / drug effects
  • Down-Regulation / physiology
  • Epilepsy / chemically induced
  • Epilepsy / pathology
  • Epilepsy / physiopathology*
  • GABA Antagonists / pharmacology
  • Guanylate Kinases
  • Hippocampus / drug effects
  • Hippocampus / pathology
  • Hippocampus / physiopathology*
  • Intracellular Signaling Peptides and Proteins / metabolism
  • Membrane Proteins / metabolism
  • Neuronal Plasticity / drug effects
  • Neuronal Plasticity / physiology*
  • Organ Culture Techniques
  • Pseudopodia / drug effects
  • Pseudopodia / pathology
  • Pseudopodia / physiology
  • Pyridazines / pharmacology
  • Rats
  • Sodium Channel Blockers / pharmacology
  • Synapses / drug effects
  • Synapses / pathology
  • Synapses / physiology*
  • Synaptic Membranes / drug effects
  • Synaptic Membranes / metabolism
  • Synaptic Membranes / pathology
  • Synaptic Transmission / drug effects
  • Synaptic Transmission / physiology

Substances

  • Disks Large Homolog 4 Protein
  • Dlg4 protein, mouse
  • GABA Antagonists
  • Intracellular Signaling Peptides and Proteins
  • Membrane Proteins
  • Pyridazines
  • Sodium Channel Blockers
  • gabazine
  • Guanylate Kinases