Immunological alterations in pregnant women with acute hepatitis E

J Gastroenterol Hepatol. 2005 Jul;20(7):1094-101. doi: 10.1111/j.1440-1746.2005.03875.x.

Abstract

Background: Infection with hepatitis E virus (HEV) is a major cause of acute viral hepatitis in several developing countries. Although usually self-limiting and benign, the disease is particularly severe among pregnant women, with mortality rates reaching 15-20%.

Methods: Immune parameters among pregnant women with acute hepatitis E (P-HEV) were investigated and compared with those in non-pregnant patients with hepatitis E (N-HEV), and healthy pregnant (PC) and non-pregnant (NPC) women.

Results: Peripheral blood mononuclear cells (PBMC) from P-HEV patients had lower lymphocyte proliferation response to phytohemagglutinin (PHA) than those in the PC and NPC groups. A positive lymphocyte proliferation response to HEV antigen (HEVAg), a mixture of eight peptides derived from HEV proteins, was observed in 7/19 (37%) P-HEV patients, 3/9 (33%) N-HEV patients and only 2/21 (10%) PC and 2/14 (14%) NPC subjects; the stimulation indices in the P-HEV group were similar to the N-HEV group and higher than the PC group. Measurement of cytokine production by PBMC in response to PHA and HEVAg showed a reduction in production of T-helper 1 (Th1) cytokines and an increase in that of Th2 cytokines in the P-HEV group. Cytokine mRNA levels showed similar changes.

Conclusion: These results show the existence of a Th2 bias in pregnant women with acute hepatitis E. The role of this Th2 bias in the greater severity of hepatitis E among pregnant women needs further investigation.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Disease
  • Adult
  • Antibodies, Viral / immunology
  • Antigens, Viral* / genetics
  • Antigens, Viral* / immunology
  • Blotting, Southern
  • Cell Proliferation
  • Cytokines / genetics
  • Cytokines / metabolism
  • Enzyme-Linked Immunosorbent Assay
  • Female
  • Hepatitis E / immunology*
  • Hepatitis E / virology
  • Humans
  • Phytohemagglutinins
  • Pregnancy
  • Pregnancy Complications, Infectious*
  • RNA, Messenger / genetics
  • RNA, Viral / genetics
  • Reverse Transcriptase Polymerase Chain Reaction
  • Th1 Cells / immunology*
  • Th1 Cells / metabolism
  • Th1 Cells / pathology
  • Th2 Cells / immunology*
  • Th2 Cells / metabolism
  • Th2 Cells / pathology

Substances

  • Antibodies, Viral
  • Antigens, Viral
  • Cytokines
  • Phytohemagglutinins
  • RNA, Messenger
  • RNA, Viral