The immune privilege of the eye: human retinal pigment epithelial cells selectively modulate T-cell activation in vitro

Curr Eye Res. 2005 May;30(5):375-83. doi: 10.1080/02713680590934120.

Abstract

Purpose: To examine the effect of human retinal pigment epithelial (RPE) cells on phytohemagglutinin (PHA) activation of T cells.

Methods: Resting peripheral blood lymphocytes (PBLs) were stimulated with PHA with or without the presence of gamma-irradiated RPE cells. Proliferation and the cell cycle profile were thereafter investigated by 3H-thymidine incorporation and flow cytometric analysis. In addition, the PBLs expression of CD69, major histocompatibility complex (MHC) class I and II, CD3, as well as the IL-2 receptor chains were evaluated by flow cytometry, and the content of IL-2 in cell culture supernatant was measured by ELISA.

Results: Human RPE cells were found to suppress PHA-induced proliferation, cyclin A, IL-2R-alpha and -gamma, and CD71 expression and decrease the production of IL-2; but RPE cells do not inhibit the PHA-induced expression of early activation markers CD69, MHC class I and II, and of cyclin D of the PBLs.

Conclusions: These results are the first to indicate that RPE cells impede generation of activated T cells by interfering with the induction of high-affinity IL-2R-alphabetagamma, IL-2 production, and the expression of CD71 and cyclin A.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, CD / metabolism
  • Antigens, Differentiation, T-Lymphocyte / metabolism
  • CD3 Complex / metabolism
  • Cell Cycle
  • Cell Line
  • Cell Proliferation
  • Coculture Techniques
  • DNA Replication
  • Enzyme-Linked Immunosorbent Assay
  • Eye / immunology*
  • Flow Cytometry
  • Histocompatibility Antigens Class I / metabolism
  • Histocompatibility Antigens Class II / metabolism
  • Humans
  • Immune System / physiology
  • Interleukin-2 Receptor alpha Subunit
  • Lectins, C-Type
  • Lymphocyte Activation*
  • Pigment Epithelium of Eye / immunology*
  • Receptors, Interleukin / metabolism
  • T-Lymphocytes / immunology*

Substances

  • Antigens, CD
  • Antigens, Differentiation, T-Lymphocyte
  • CD3 Complex
  • CD69 antigen
  • Histocompatibility Antigens Class I
  • Histocompatibility Antigens Class II
  • IL2RA protein, human
  • Interleukin-2 Receptor alpha Subunit
  • Lectins, C-Type
  • Receptors, Interleukin