Is M129V of PRNP gene associated with Alzheimer's disease? A case-control study and a meta-analysis

Neurobiol Aging. 2006 May;27(5):770.e1-770.e5. doi: 10.1016/j.neurobiolaging.2005.05.025. Epub 2005 Aug 15.

Abstract

The methionine/valine (M/V) polymorphism at codon 129 within the prion protein gene (PRNP) represents a known risk factor for Creutzfeldt-Jakob disease (CJD). Few authors reported also the effects of this polymorphism on the risk of Alzheimer's disease (AD), although with controversial results. To better clarify this issue, we performed a novel case-control study and a meta-analysis of published association studies between PRNP and AD. Our findings argue against PRNP as a susceptibility gene for developing AD in the Italian population but support the hypothesis that the V allele influences cognitive performances. The meta-analysis, revealed that Caucasian subjects homozygous at codon 129 had a 1.3-fold increased risk [95% CI: 1.0-1.6, p = 0.05] of developing AD compared to heterozygous individuals. We also observed that MM genotype and M allele represent a risk factor for AD, independently from the ethnic background, providing a significant but modest association between this polymorphism and AD.

Publication types

  • Meta-Analysis
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Alleles
  • Alzheimer Disease / epidemiology
  • Alzheimer Disease / genetics*
  • Alzheimer Disease / psychology
  • Amyloid / genetics*
  • Apolipoproteins E / genetics
  • Case-Control Studies
  • Cognition / physiology
  • DNA / genetics
  • Female
  • Gene Frequency
  • Genotype
  • Humans
  • Italy / epidemiology
  • Male
  • Neuropsychological Tests
  • Polymorphism, Genetic / genetics
  • Prion Proteins
  • Prions
  • Protein Precursors / genetics*

Substances

  • Amyloid
  • Apolipoproteins E
  • PRNP protein, human
  • Prion Proteins
  • Prions
  • Protein Precursors
  • DNA