A short-incubation reporter-gene assay for high-throughput screening of estrogen receptor-alpha antagonists

Assay Drug Dev Technol. 2005 Aug;3(4):393-400. doi: 10.1089/adt.2005.3.393.

Abstract

Estrogen receptor (ER) alpha and beta are ligand-activated nuclear transcription factors that mediate the effects of the steroid hormone 17beta-estradiol. Tissue-selective ER modulators have been developed for the treatment of a variety of diseases, including osteoporosis and hormone-dependent breast cancer. Second- and third-generation selective ER modulators are in development, with the goal of reducing toxicity and improving tissue-selective efficacy. Novel tissue-selective and ERsubtype specific ligands may have the potential of providing a new paradigm for maintaining the health of women. The traditional cell-based screening assays for nuclear receptors require 16-18 h of incubation, which limits the assay miniaturization for ultra-high-throughput screening. We have developed a new cell-based ERalpha transactivation assay for the screening of ERalpha-specific antagonists with only 4 h of incubation time. The assay was optimized and used for a fully automated ultrahigh-throughput screen in 3,456-well nanoplate format. The screening throughput was 250,000-300,000 compounds per day, and a number of valuable leads were identified.

MeSH terms

  • Animals
  • CHO Cells
  • Cricetinae
  • Cricetulus
  • Dose-Response Relationship, Drug
  • Drug Evaluation, Preclinical / methods
  • Estradiol / analogs & derivatives
  • Estradiol / pharmacology
  • Estrogen Antagonists / analysis
  • Estrogen Antagonists / pharmacology*
  • Estrogen Receptor alpha / antagonists & inhibitors*
  • Estrogen Receptor alpha / genetics
  • Estrogen Receptor alpha / metabolism
  • Fluorescence Resonance Energy Transfer / methods
  • Fulvestrant
  • Genetic Vectors
  • Raloxifene Hydrochloride / pharmacology
  • Recombinant Fusion Proteins / metabolism
  • Selective Estrogen Receptor Modulators / pharmacology
  • Tamoxifen / pharmacology
  • Transcription Factors / genetics
  • Transcription Factors / metabolism
  • Transcriptional Activation
  • Transfection
  • beta-Lactamases / genetics
  • beta-Lactamases / metabolism

Substances

  • Estrogen Antagonists
  • Estrogen Receptor alpha
  • Recombinant Fusion Proteins
  • Selective Estrogen Receptor Modulators
  • Transcription Factors
  • Tamoxifen
  • Fulvestrant
  • Raloxifene Hydrochloride
  • Estradiol
  • beta-Lactamases