Differentiation of circulating endothelial progenitor cells to a cardiomyogenic phenotype depends on E-cadherin

FEBS Lett. 2005 Nov 7;579(27):6060-6. doi: 10.1016/j.febslet.2005.09.071. Epub 2005 Oct 6.

Abstract

Progenitor cells may contribute to cardiac regeneration. Here, we investigated the role of cadherins and integrins for differentiation of human adult circulating endothelial progenitor cells (EPCs) into cardiomyocytes (CM) in a co-culture system. N- and E-cadherin were expressed in EPCs and were localized at the interface between EPCs and CM. Incubation of a blocking antibody against E-cadherin reduced the expression of CM marker protein in EPCs. Blocking antibodies against N- or P-cadherin or the beta1- and beta2-integrins were not effective. These data suggested that cell-to-cell communication mediated by E-cadherin contributes to the acquirement of a cardiomyogenic phenotype of human endothelial progenitor cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antibodies, Blocking / pharmacology
  • CD18 Antigens / drug effects
  • CD18 Antigens / immunology
  • CD18 Antigens / metabolism
  • Cadherins / drug effects
  • Cadherins / immunology
  • Cadherins / metabolism*
  • Cell Communication
  • Cell Differentiation
  • Cells, Cultured
  • Coculture Techniques
  • Endothelial Cells / cytology*
  • Endothelial Cells / physiology
  • Humans
  • Integrin beta1 / drug effects
  • Integrin beta1 / immunology
  • Integrin beta1 / metabolism
  • Myocytes, Cardiac / cytology*
  • Phenotype
  • Stem Cells / cytology*
  • Stem Cells / physiology

Substances

  • Antibodies, Blocking
  • CD18 Antigens
  • Cadherins
  • Integrin beta1