Aging affect the anti-tumor potential of dendritic cell vaccination, but it can be overcome by co-stimulation with anti-OX40 or anti-4-1BB

Exp Gerontol. 2006 Jan;41(1):78-84. doi: 10.1016/j.exger.2005.10.002. Epub 2005 Nov 14.

Abstract

It has been well established that there is a decline in immune function with age resulting in a diminished capacity to respond to infections or tumors. Although many studies have demonstrated the efficacy of autologous dendritic cells (DC) vaccines in stimulating an anti-tumor immune response in the young, almost none of these reports consider the effect that aging has on the immune system or test whether DC-vaccination is effective in old hosts. In this study we compared the efficacy of DC-vaccination in young and old mice. Our results showed that DC-vaccination in young animals induced an anti-tumor response resulting in approximately 60% tumor growth inhibition, while minimal protection was observed in old animals. DC vaccination plus rIL-2 further enhanced the anti-tumor response in young animals (approximately 70-75% inhibition), while ineffective in old animals. In contrast, co-administration of anti-OX-40 or anti-4-1BB mAbs vigorously enhanced the anti-tumor immune response in both young (approximately 85-90% inhibition) and old mice (approximately 70-75% inhibition). Our data indicate that although old mice have a decline in immune function, they have the capacity to develop strong anti-tumor responses as long as they are provided with efficient co-stimulation.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Adenocarcinoma / pathology
  • Adenocarcinoma / prevention & control*
  • Aging / immunology*
  • Animals
  • Antibodies, Monoclonal / immunology
  • Antigens, CD / immunology
  • Apoptosis / immunology
  • Cancer Vaccines / immunology*
  • Cytotoxicity, Immunologic
  • Dendritic Cells / transplantation*
  • Immunization
  • Interleukin-2 / immunology
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Neoplasm Transplantation
  • Prostatic Neoplasms / pathology
  • Prostatic Neoplasms / prevention & control*
  • Receptors, Nerve Growth Factor / immunology
  • Receptors, OX40
  • Receptors, Tumor Necrosis Factor / immunology
  • Recombinant Proteins / immunology
  • Tumor Necrosis Factor Receptor Superfamily, Member 9

Substances

  • Antibodies, Monoclonal
  • Antigens, CD
  • Cancer Vaccines
  • Interleukin-2
  • Receptors, Nerve Growth Factor
  • Receptors, OX40
  • Receptors, Tumor Necrosis Factor
  • Recombinant Proteins
  • Tnfrsf4 protein, mouse
  • Tnfrsf9 protein, mouse
  • Tumor Necrosis Factor Receptor Superfamily, Member 9