V617F mutation in JAK2 is associated with poorer survival in idiopathic myelofibrosis

Blood. 2006 Mar 1;107(5):2098-100. doi: 10.1182/blood-2005-08-3395. Epub 2005 Nov 17.

Abstract

Most patients with polycythemia vera and half with idiopathic myelofibrosis and essential thrombocythemia have an acquired V617F mutation in JAK2. Using sensitive polymerase chain reaction (PCR)-based methods, we genotyped 152 patients with idiopathic myelofibrosis to establish whether there were differences in presentation and outcome between those with and those without the mutation. Patients positive for V617F had higher neutrophil and white cell counts (P = .02) than did patients negative for V617F, but other diagnostic features were comparable between the 2 groups. Patients positive for V617F were less likely to require blood transfusion during follow-up (P = .03). Despite this, patients positive for V617F had poorer overall survival, even after correction for confounding factors (P = .01).

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Amino Acid Substitution*
  • Blood Transfusion
  • Disease-Free Survival
  • Female
  • Follow-Up Studies
  • Humans
  • Janus Kinase 2
  • Male
  • Middle Aged
  • Point Mutation*
  • Polycythemia Vera / genetics*
  • Polycythemia Vera / mortality
  • Polycythemia Vera / therapy
  • Primary Myelofibrosis / complications
  • Primary Myelofibrosis / genetics*
  • Primary Myelofibrosis / mortality
  • Primary Myelofibrosis / therapy
  • Protein-Tyrosine Kinases / genetics*
  • Proto-Oncogene Proteins / genetics*
  • Thrombocythemia, Essential / complications
  • Thrombocythemia, Essential / genetics*
  • Thrombocythemia, Essential / mortality
  • Thrombocythemia, Essential / therapy

Substances

  • Proto-Oncogene Proteins
  • Protein-Tyrosine Kinases
  • JAK2 protein, human
  • Janus Kinase 2