Cutting edge: murine CD59a modulates antiviral CD4+ T cell activity in a complement-independent manner

J Immunol. 2005 Dec 1;175(11):7098-102. doi: 10.4049/jimmunol.175.11.7098.

Abstract

CD59 blocks formation of the membrane attack complex of complement by inhibiting binding of C9 to the C5b-8 complex. To investigate a role for CD59 in promoting T cell responses, we compared T cell activation in CD59a-deficient (Cd59a-/-) and wild-type (WT) mice after in vitro stimulation and after infection with rVV. Virus-specific CD4+ T cell responses were significantly enhanced in Cd59a-/- mice compared with WT mice. Similarly, Cd59a-/- T cells responded more vigorously to in vitro stimulation with CD3-specific Abs compared with WT mice. This effect of CD59a on T cell proliferation was found to be complement-independent. Collectively, these results demonstrate that CD59a down-modulates CD4+ T cell activity in vitro and in vivo, thereby revealing another link between complement regulators and T cell activation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Blotting, Western
  • CD4-Positive T-Lymphocytes / immunology*
  • CD4-Positive T-Lymphocytes / virology*
  • CD59 Antigens / immunology*
  • Cell Proliferation
  • Complement System Proteins / immunology*
  • Immunoprecipitation
  • Lymphocyte Activation / immunology
  • Lymphocytic choriomeningitis virus / immunology
  • Mice
  • Vaccinia virus / immunology
  • Viral Proteins / immunology

Substances

  • CD59 Antigens
  • Viral Proteins
  • Complement System Proteins