Cutting edge: the development of IL-4-producing B cells (B effector 2 cells) is controlled by IL-4, IL-4 receptor alpha, and Th2 cells

J Immunol. 2005 Dec 1;175(11):7103-7. doi: 10.4049/jimmunol.175.11.7103.

Abstract

Although IL-4-producing B cells (B effector 2 cells) are found following infection and immunization, the signals regulating IL-4 production by Be2 cells are unknown. We show that culturing naive B cells with Th2 cells induces up-regulation of IL-4 in the B cells with a concomitant down-regulation of T-bet, IL-12Rbeta2, and IFN-gamma. Up-regulation of IL-4 in the Be2 cells is dependent on both T cells and IL-4 as IL-4Ralpha-deficient B cells primed with Th2 cells did not transcribe IL-4, and B cells primed in the presence of IL-4-deficient Th2 cells produced IFN-gamma instead of IL-4. Likewise, the in vivo development of IL-4-expressing B cells in a nematode infection model was dependent on both T cells and IL-4Ralpha-mediated signals. Thus, the differentiation of naive B cells into IL-4-expressing Be2 cells is regulated by a combination of T cell-dependent signals and the cytokine environment and this process is critically dependent upon the IL-4/IL-4R signaling pathway.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Animals
  • B-Lymphocytes / cytology*
  • B-Lymphocytes / immunology*
  • B-Lymphocytes / metabolism
  • Cell Differentiation / immunology
  • Cells, Cultured
  • Interferon-gamma / biosynthesis
  • Interferon-gamma / immunology
  • Interleukin-4 / biosynthesis
  • Interleukin-4 / immunology*
  • Mice
  • Nematospiroides dubius / immunology
  • Receptors, Cell Surface / immunology*
  • Receptors, Interleukin / biosynthesis
  • Receptors, Interleukin / immunology
  • Receptors, Interleukin-12
  • Reverse Transcriptase Polymerase Chain Reaction
  • Th2 Cells / immunology*

Substances

  • Il4ra protein, mouse
  • Receptors, Cell Surface
  • Receptors, Interleukin
  • Receptors, Interleukin-12
  • Interleukin-4
  • Interferon-gamma