Leptin-mediated inhibition of the insulin-stimulated increase in fatty acid uptake in differentiated 3T3-L1 adipocytes

Metabolism. 2006 Jan;55(1):8-12. doi: 10.1016/j.metabol.2005.06.013.

Abstract

The effects of insulin and leptin on fatty acid uptake in differentiated (adipocytes) and undifferentiated 3T3-L1 cells were investigated. It was demonstrated that in undifferentiated 3T3-L1 cells, insulin and leptin have no effect on fatty acid uptake. In differentiated 3T3-L1 adipocytes, insulin had a concentration-dependent stimulatory effect on fatty acid uptake, whereas leptin on its own had no effect. Leptin, when coincubated with 10 nmol/L insulin, resulted in a concentration-dependent inhibition of the insulin-stimulated fatty acid uptake in differentiated 3T3-L1 cells. These results indicate that leptin has a direct inhibitory effect on the stimulation of fatty acid uptake by insulin in differentiated murine adipocytes.

MeSH terms

  • 3T3 Cells
  • Adipocytes / drug effects
  • Adipocytes / metabolism*
  • Animals
  • Cell Differentiation
  • DNA / biosynthesis
  • DNA / genetics
  • Fatty Acids / metabolism*
  • Hypoglycemic Agents / antagonists & inhibitors*
  • Hypoglycemic Agents / pharmacology*
  • Insulin / pharmacology*
  • Leptin / pharmacology*
  • Mice
  • RNA / biosynthesis
  • RNA / genetics
  • Reverse Transcriptase Polymerase Chain Reaction
  • Spectrometry, Fluorescence

Substances

  • Fatty Acids
  • Hypoglycemic Agents
  • Insulin
  • Leptin
  • RNA
  • DNA