Interleukin 6 gene transcripts are expressed in atherosclerotic lesions of genetically hyperlipidemic rabbits

Atherosclerosis. 1992 Feb;92(2-3):213-8. doi: 10.1016/0021-9150(92)90280-t.

Abstract

We have investigated the involvement of interleukin 6 (IL-6), a growth-regulatory molecule for vascular smooth muscle cells (SMC), in the development of atherosclerotic lesions of Watanabe heritable hyperlipidemic (WHHL) rabbits. In in situ hybridization analysis, quite low levels of IL-6 mRNA were expressed in 'quiescent' SMC cultured from WHHL rabbits; however, high levels of IL-6 mRNA were induced in SMC exposed to 10% fetal bovine serum (FBS), suggesting that growth-stimulated SMC themselves can synthesize IL-6. In in vivo WHHL aortae, transcripts for the IL-6 gene were clearly observed in the fibrous plaques. These findings support the premise that IL-6 is an important autocrine and/or paracrine regulator of SMC proliferation and of pathogenesis of atherosclerosis in this animal model.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Arteriosclerosis / genetics*
  • Arteriosclerosis / metabolism
  • Disease Models, Animal
  • Gene Expression
  • Hyperlipidemias / genetics*
  • Hyperlipidemias / metabolism
  • Interleukin-6 / genetics*
  • Male
  • Muscle, Smooth, Vascular / metabolism
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Rabbits
  • Transcription, Genetic

Substances

  • Interleukin-6
  • RNA, Messenger