Abstract
Human immunodeficiency virus type 1 (HIV-1) uses a chemokine receptor, usually CXCR4 or CCR5, for entry into the target cells. Here, we used a chemical biology approach to demonstrate that binding and signaling domains in CXCR4 are possibly distinct and separate, as the new analogue, D(1-10)-vMIP-II-(9-68)-SDF-1alpha (RCP222), could not activate CXCR4 despite the fact that its binding activity was comparable to that of stromal cell-derived factor (SDF)-1alpha, the only natural ligand of CXCR4.
Publication types
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Research Support, N.I.H., Extramural
MeSH terms
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Amino Acid Sequence
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Amino Acids / chemistry*
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Anti-HIV Agents / chemistry*
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Anti-HIV Agents / pharmacology
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Binding, Competitive
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Calcium / metabolism
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Cell Line
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Chemokine CXCL12
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Chemokines / chemistry
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Chemokines / metabolism*
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Chemokines / pharmacology
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Chemokines, CXC / metabolism
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HIV-1 / drug effects
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Humans
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Ligands
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Molecular Conformation
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Molecular Sequence Data
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Protein Structure, Tertiary
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Radioligand Assay
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Receptors, CXCR4 / agonists
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Receptors, CXCR4 / metabolism
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Receptors, CXCR4 / physiology*
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Signal Transduction
Substances
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Amino Acids
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Anti-HIV Agents
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CXCL12 protein, human
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Chemokine CXCL12
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Chemokines
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Chemokines, CXC
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D(1-10)-vMIP-II-(9-68)-SDF-1alpha
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Ligands
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Receptors, CXCR4
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vMIP-II
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Calcium