Effects of gastric bypass on the GH/IGF-I axis in severe obesity--and a comparison with GH deficiency

Eur J Endocrinol. 2006 Jan;154(1):53-9. doi: 10.1530/eje.1.02069.

Abstract

Objective: Overfeeding suppresses GH secretion and makes evaluation of a suspected GH deficiency (GHD) difficult. In normal weight subjects, gender is known to influence GH concentrations, which is most apparent in the ambulatory, morning-fasted state. In this study, we examined the GH/IGF-I axis in obese men and women and the effect of surgically induced weight loss.

Design: Sixty-three subjects (body mass index (BMI) 45 +/- 6 kg/m2; 54 women, 9 men) were studied prior to, and 6 and 12 months following Roux-en-Y gastric bypass (RYGBP) surgery. Fifty-four patients with classic GHD (BMI 27 +/- 6 kg/m2; 35 men, 19 women) were included for comparison.

Methods: Hormones were analysed in fasting morning serum samples.

Results: RYGBP resulted in a decreased BMI to 35 +/- kg/m2 at 6 months and 32 +/- 6 kg/m2 at 12 months. GH and IGF-I increased at 6 months in the women and at 12 months in both sexes by > or = 300 and 11% respectively. Prior to RYGBP, GH concentrations were low in the obese men and similar to those of GHD men (mean 0.09 mU/l). Obese women had tenfold higher values than obese men and sevenfold higher than GHD women. IGF-I levels were in the low reference range in the obese and below -2 S.D. for age in 13%.

Conclusions: Surgically induced weight loss partially restores GH secretion. Despite a marked suppression of GH values, a gender influence is maintained in severe obesity. In obese women, single morning GH and IGF-I values seem sufficient to exclude a suspicion of classic GHD.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Body Mass Index
  • Female
  • Gastric Bypass*
  • Glucose / metabolism
  • Homeostasis
  • Human Growth Hormone / deficiency*
  • Human Growth Hormone / metabolism*
  • Humans
  • Insulin-Like Growth Factor I / metabolism*
  • Male
  • Middle Aged
  • Obesity / physiopathology*
  • Obesity / surgery*

Substances

  • Human Growth Hormone
  • Insulin-Like Growth Factor I
  • Glucose