Effects of N-acetylcysteine on myocardial ischemia-reperfusion injury in bypass surgery

Heart Vessels. 2006 Jan;21(1):42-7. doi: 10.1007/s00380-005-0873-1.

Abstract

Myocardial ischemia-reperfusion injury may complicate coronary artery bypass grafting (CABG) operations. N-Acetylcysteine (NAC) had antioxidant and microcirculatory effects, and inhibits neutrophil aggregation. The aim of this study was to determine the effects of NAC in limiting myocardial ischemia-reperfusion injury in CABG operations. Twenty patients undergoing elective coronary bypass operation with cardiopulmonary bypass were enrolled and randomly assigned to two groups: a control group operated with a routine CABG protocol, and one where NAC was administered intravenously during the operation (NAC group). Blood samples from coronary sinus for tumor necrosis factor-alpha assay, myocardial biopsy specimens for chemiluminescent luminol, and lucigenin measurements of reactive oxygen species were taken. The luminol (specific for (*)OH, H(2)O(2), and HOCl(-) radicals) and lucigenin (specific for O(2) (*-)) levels and the difference ratios after reperfusion were significantly lower in the NAC group. Tumor necrosis factor-alpha levels increased in the control group but, in contrast, a significant decrease was detected in the NAC group (P < 0.01). Creatine kinase-MB levels at 6 and 12 hours were significantly lower in the NAC group (P = 0.02). N-Acetylcysteine has potential effects to limit ischemia reperfusion injury during CABG operations. We believe that its effects on clinical outcome may be more apparent in patients prone to ischemia-reperfusion injury.

Publication types

  • Randomized Controlled Trial

MeSH terms

  • Acetylcysteine / therapeutic use*
  • Acridines
  • Aged
  • Biomarkers / blood
  • Cardiopulmonary Bypass
  • Coronary Artery Bypass*
  • Coronary Artery Disease / surgery
  • Creatine Kinase, MB Form / blood
  • Creatine Kinase, MB Form / drug effects
  • Female
  • Follow-Up Studies
  • Free Radical Scavengers / therapeutic use*
  • Humans
  • Indicators and Reagents
  • Inflammation Mediators / blood
  • Luminescent Measurements
  • Luminol
  • Male
  • Middle Aged
  • Myocardial Reperfusion Injury / blood
  • Myocardial Reperfusion Injury / prevention & control*
  • Oxidative Stress / drug effects
  • Reactive Oxygen Species / blood
  • Research Design
  • Treatment Outcome
  • Tumor Necrosis Factor-alpha / blood
  • Tumor Necrosis Factor-alpha / drug effects

Substances

  • Acridines
  • Biomarkers
  • Free Radical Scavengers
  • Indicators and Reagents
  • Inflammation Mediators
  • Reactive Oxygen Species
  • Tumor Necrosis Factor-alpha
  • 10,10'-dimethyl-9,9'-biacridinium
  • Luminol
  • Creatine Kinase, MB Form
  • Acetylcysteine