Effect of nifedipine on cyclosporine A-induced nephrotoxicity, urinary endothelin excretion and renal endothelin receptor number

Eur J Pharmacol. 1991 Feb 26;194(1):115-7. doi: 10.1016/0014-2999(91)90132-a.

Abstract

The aim of the present study was to determine the effect of a calcium channel blocker on renal function, urinary endothelin excretion and endothelin receptor number in rats. Administration of cyclosporine resulted in a significant impairment of renal function when measured by either [14C]inulin or 24 h creatinine clearances. This nephrotoxicity was associated with statistically significant (P less than 0.05) increases in urinary endothelin excretion and renal endothelin receptor number. Treatment with nifedipine attenuated cyclosporine A-induced renal dysfunction and reduced urinary endothelin excretion. The data provide further evidence of a role for endothelin in cyclosporine A-induced nephrotoxicity.

MeSH terms

  • Animals
  • Blood Urea Nitrogen
  • Creatinine / blood
  • Cyclosporins / toxicity*
  • Endothelins / urine*
  • Glomerular Filtration Rate / drug effects
  • In Vitro Techniques
  • Kidney / drug effects
  • Kidney / metabolism*
  • Kidney Diseases / chemically induced
  • Kidney Diseases / physiopathology
  • Kidney Diseases / prevention & control*
  • Male
  • Membranes / drug effects
  • Membranes / metabolism
  • Nifedipine / pharmacology*
  • Rats
  • Rats, Inbred Strains
  • Receptors, Cell Surface / drug effects
  • Receptors, Cell Surface / metabolism*
  • Receptors, Endothelin

Substances

  • Cyclosporins
  • Endothelins
  • Receptors, Cell Surface
  • Receptors, Endothelin
  • Creatinine
  • Nifedipine