Association of Ki-ras mutation with differentiation and tumor-formation pathways in colorectal carcinoma

Int J Cancer. 1991 Sep 9;49(2):220-3. doi: 10.1002/ijc.2910490213.

Abstract

The occurrence of a point mutation on the 12th and 13th codons of the Ki-ras oncogene has been investigated in 99 colorectal carcinomas in relation to 3 histological parameters: extent of differentiation, occurrence of a mucinous component within the tumor, and presence of peripheral adenomatous polyp remnants. The mutation frequency increased with each parameter: from 13% (2/15) to 44% (37/84) with differentiation, from 33% (26/79) to 65% (13/20) with mucinous character, and from 27% (15/56) to 56% (24/43) with the presence of polyp remnants. The frequency was highest in well-differentiated mucinous tumors with adenomatous remnants 83% (5/6). We suggest that Ki-ras mutation is preferentially involved in carcinomas that have developed from adenoma and that the mutation preserves differentiation and mucin secretion in these cancer cells.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenocarcinoma, Mucinous / genetics
  • Adenocarcinoma, Mucinous / pathology
  • Base Sequence
  • Codon / genetics*
  • Colonic Polyps / genetics
  • Colonic Polyps / pathology
  • Colorectal Neoplasms / genetics*
  • Colorectal Neoplasms / pathology
  • Genes, ras / genetics*
  • Humans
  • Middle Aged
  • Molecular Sequence Data
  • Mutation / genetics*

Substances

  • Codon