The 460Trp allele of alpha-adducin increases carotid intima-media thickness in young adult males

J Hypertens. 2006 Apr;24(4):697-703. doi: 10.1097/01.hjh.0000217852.89745.dc.

Abstract

Background: The 460Trp allele of the alpha-adducin gene (ADD1), which is involved in a form of salt-sensitive hypertension, has been associated with patterns of target organ damage.

Objectives: As carotid artery intima-media thickness (IMT) largely depends upon unknown genetic factors, besides being associated to conventional risk factors, we tested the association of the 460Trp allele of ADD1 with IMT in a well-characterized sample of young healthy normotensive subjects, to assess the role of ADD1 polymorphism without overlapping effects of age or already elevated blood pressure.

Methods: Anthropometric measurements, blood pressure (BP), and carotid artery wall IMT (high-resolution sonography and digitalized morphometry) were obtained in 420 healthy normotensive Caucasian university students. Genotypes for ADD1 were detected by automated genomic polymerase chain reaction (PCR).

Results: ADD1 genotypes were evenly distributed between genders. IMT was significantly larger in carriers of the 460Trp allele of ADD1, while a significant gender x ADD1 interaction (P = 0.02) demonstrated that IMT was increased only in males carrying the 460Trp allele (P < 0.001). No significant association was found in females.

Conclusions: The 460Trp allele of ADD1 contributes substantially to increase carotid IMT, in a male hormonal milieu only, at least in the young age range.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Amino Acid Substitution
  • Calmodulin-Binding Proteins / genetics*
  • Carotid Arteries / diagnostic imaging
  • Carotid Arteries / pathology*
  • Female
  • Gene Frequency
  • Genotype
  • Humans
  • Male
  • Polymorphism, Genetic*
  • Sex Factors
  • Tunica Intima / diagnostic imaging
  • Tunica Intima / pathology*
  • Tunica Media / diagnostic imaging
  • Tunica Media / pathology*
  • Ultrasonography

Substances

  • Calmodulin-Binding Proteins
  • adducin