Normal lymphocyte responsiveness to lectins but impaired sensitivity to in vitro glucocorticoids in major depression

J Affect Disord. 1991 Aug;22(4):241-8. doi: 10.1016/0165-0327(91)90070-9.

Abstract

The mitogen-induced lymphocyte proliferative response and its sensitivity to in vitro (10(-10)-10(-6) M) dexamethasone (DEX) administration were investigated in 12 severely depressed patients and 13 healthy controls. Patients with major depressive disorder exhibited no impairment of lectin-induced blastogenesis, but a significantly weaker suppressive effect of in vitro DEX on 1.0 microgram/ml phytohemagglutinin A-induced proliferation. The inhibitory potency of in vitro DEX was inversely correlated with in vivo adrenal cortical hormone levels at 4.00 p.m. These effects were not observed with pokeweed mitogen- and concanavalin A-stimulated cells. There were no correlations with age, weight, sex or severity of depression. These results do not support the hypothesis of a primarily impaired cell-mediated immunity, but might be indicative of reduced glucocorticoid receptor sensitivity in major depressive disorder.

MeSH terms

  • Adrenocorticotropic Hormone / blood
  • Adult
  • Cells, Cultured
  • Depressive Disorder / immunology*
  • Dexamethasone / pharmacology*
  • Female
  • Humans
  • Hydrocortisone / blood
  • Immune Tolerance / drug effects
  • Immune Tolerance / immunology
  • Immunity, Cellular / drug effects
  • Immunity, Cellular / immunology
  • Lymphocyte Activation / drug effects*
  • Lymphocyte Activation / immunology*
  • Male
  • Middle Aged
  • Phytohemagglutinins / immunology*

Substances

  • Phytohemagglutinins
  • Dexamethasone
  • Adrenocorticotropic Hormone
  • Hydrocortisone