Regulation of human neurotropic JCV in colon cancer cells

Anticancer Res. 2006 Mar-Apr;26(2A):833-41.

Abstract

Background: Recently, the genome of the human polyomavirus, JC (JCV), and expression of its early and late regulatory proteins (T-antigen and agnoprotein) have been demonstrated in neoplastic cells of colonic cancer cases.

Materials and methods: Regulation of JCV was investigated in a human colon cancer cell line (SW480) and compared to a human glioblastoma cell line (U87-MG) that is permissive for JCV replication.

Results: SW480 cells supported basal transcription of both early and late JCV promoters. The expression of TCF-4, a component of Wnt signaling, modulated JCV transcription in a cell type-specific manner. Both TCF-4 and T-antigen bound to the JCV promoter region and bound to each other. In addition, the expression of TCF-4 caused a decrease in the ability of the T-antigen to stimulate viral DNA replication in U87-MG cells.

Conclusion: Wnt pathway signaling proteins and T-antigen interact to regulate JCV in colonic epithelial cells.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Antigens, Viral, Tumor / genetics
  • Antigens, Viral, Tumor / metabolism
  • Antigens, Viral, Tumor / physiology
  • Cell Line, Tumor
  • Colonic Neoplasms / genetics
  • Colonic Neoplasms / virology*
  • DNA, Viral / genetics
  • DNA, Viral / metabolism
  • Glioblastoma / genetics
  • Glioblastoma / virology
  • Humans
  • JC Virus / genetics
  • JC Virus / immunology
  • JC Virus / physiology*
  • Promoter Regions, Genetic
  • Signal Transduction
  • TCF Transcription Factors / genetics
  • TCF Transcription Factors / physiology
  • Transcription, Genetic
  • Transfection
  • Virus Replication
  • Wnt Proteins / genetics
  • Wnt Proteins / physiology

Substances

  • Antigens, Viral, Tumor
  • DNA, Viral
  • TCF Transcription Factors
  • Wnt Proteins