Differential regulation of jun family gene expression by the tumor promoter okadaic acid

New Biol. 1991 Oct;3(10):977-86.

Abstract

Phosphorylation events are major regulatory mechanisms of signal transduction pathways that control cell growth and differentiation. The potential involvement of serine/threonine-specific phosphoprotein phosphatases in pathways that regulate gene expression was analyzed. By use of okadaic acid, an inhibitor of protein phosphatases 1 (PP-1) and 2A (PP-2A), we present evidence that expression of distinct members of the jun family of genes, c-jun, junB, and junD, are regulated differentially by serine/threonine-specific phosphoprotein phosphatases. Treatment of cells with okadaic acid induces the expression of junB, and to a lesser extent c-jun, but has only a marginal effect on junD expression. This induction involves transcriptional as well as post-transcriptional mechanisms. An analysis of defined elements in different promoters suggests that serine/threonine phosphoprotein phosphatases are involved in the regulation of the c-jun and the collagenase 12-O-tetradecanoyl phorbol-13-acetate (TPA) response element (TRE) as well as the c-fos serum response element (SRE). Since inhibition of PP-1 and PP-2A leads to increased proto-oncogene expression, our results further support the view that certain protein phosphatases might act as negative regulators of growth.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • 3T3 Cells / drug effects
  • 3T3 Cells / metabolism
  • Animals
  • Base Sequence
  • Carcinogens / pharmacology*
  • DNA-Binding Proteins
  • Drug Synergism
  • Ethers, Cyclic / pharmacology*
  • Gene Expression Regulation / drug effects*
  • Mice
  • Microbial Collagenase / genetics
  • Molecular Sequence Data
  • Multigene Family*
  • Okadaic Acid
  • Phosphoprotein Phosphatases / antagonists & inhibitors
  • Phosphoprotein Phosphatases / physiology*
  • Phosphorylation
  • Promoter Regions, Genetic
  • Protein Processing, Post-Translational / drug effects
  • Proto-Oncogene Proteins c-fos / biosynthesis
  • Proto-Oncogene Proteins c-fos / genetics
  • Proto-Oncogene Proteins c-jun / biosynthesis
  • Proto-Oncogene Proteins c-jun / genetics*
  • Proto-Oncogenes / drug effects*
  • Recombinant Fusion Proteins / biosynthesis
  • Regulatory Sequences, Nucleic Acid
  • Signal Transduction
  • Tetradecanoylphorbol Acetate / pharmacology

Substances

  • Carcinogens
  • DNA-Binding Proteins
  • Ethers, Cyclic
  • Proto-Oncogene Proteins c-fos
  • Proto-Oncogene Proteins c-jun
  • Recombinant Fusion Proteins
  • Okadaic Acid
  • Phosphoprotein Phosphatases
  • Microbial Collagenase
  • Tetradecanoylphorbol Acetate